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Jia-Jin Yang
Articles in Scholarly Journals [Incomplete List]
ANCA patients have T cells responsive to complementary PR-3 antigen
Kidney International, Article ID ki2008309, 2008
A study of conformational restraints on reactivity of human PR3-specific autoantibodies (ANCA) facilitated through protein folding manipulations of a new recombinant proteinase 3 protein
Autoimmunity, vol. 40, no. 7, pp. 503–511, 2007
Circumvention of Normal Constraints on Granule Protein Gene Expression in Peripheral Blood Neutrophils and Monocytes of Patients with Antineutrophil Cytoplasmic Autoantibody-Associated Glomerulonephritis
Journal of the American Society of Nephrology, vol. 15, no. 8, pp. 2103–2114, 2004
Increased expression of the secretory leukocyte proteinase inhibitor in Wegener's granulomatosis
Clinical and Experimental Immunology, vol. 131, no. 1, pp. 190–196, 2003
Novel Effects of Neutrophil-Derived Proteinase 3 and Elastase on the Vascular Endothelium Involve In Vivo Cleavage of NF- B and Proapoptotic Changes in JNK, ERK, and p38 MAPK Signaling Pathways
Journal of the American Society of Nephrology, vol. 13, no. 12, pp. 2840–2849, 2002
Expression profile of leukocyte genes activated by anti-neutrophil cytoplasmic autoantibodies (ANCA)
Kidney International, vol. 62, no. 5, pp. 1638–1649, 2002
ANCA antigens, proteinase 3 and myeloperoxidase, are not expressed in endothelial cells1
Kidney International, vol. 57, no. 5, pp. 1981–1990, 2000
Target antigens for anti-neutrophil cytoplasmic autoantibodies (ANCA) are on the surface of primed and apoptotic but not unstimulated neutrophils
Clinical and Experimental Immunology, vol. 121, no. 1, pp. 165–172, 2000
Are ANCA pathogenetic? Pros and cons
Nephrology, vol. 3, no. s2, pp. s780–s782, 1997
Primary Renovascular Effects of Erythromycin in the Rat: Relationship to Cyclosporine Nephrotoxicity
Renal Failure, vol. 12, no. 4, pp. 241–248, 1990