Ali A. Khraibi

Ali A. Khraibi has been a Professor of physiology at the Department of Physiological Sciences of Eastern Virginia Medical School, USA, since April 2001. He received his B.S. degree in 1978 and his M.S. degree in 1980 in civil engineering from the University of Mississippi, USA. He then received his Ph.D. degree in 1984 in physiology and biomedical engineering from the University of Mississippi School of Medicine, USA. For his dissertation, Dr. Khraibi received the Robert A. Mahaffey, Jr. Memorial Award for research potential in physiology and biophysics. Dr. Khraibi trained in cardiovascular, microcirculation, and renal physiology at Wake Forest University School of Medicine (1984–1986) in Winston Salem, North Carolina, USA, and at the Mayo Clinic (1986–1990) in Rochester, Minnesota, USA. He then joined the faculty at the Mayo Clinic College of Medicine where he became an Assistant Professor (1990) and then an Associate Professor (1995) in the Department of Physiology and Biophysics. In 1996, he became interested in studying cardiovascular and renal adaptations to pregnancy and accepted a faculty position as an Associate Professor in the Department of Obstetrics and Gynecology, the University of Illinois at Chicago, USA, where he stayed until 2001. Dr. Khraibi received the Young Investigator Award in Regulatory and Integrative Physiology of the American Physiological Society in 1995 and The American Society of Hypertension/Hoechst Marion Roussel Young Scholar Award in 1998. His research interests focus broadly on the role of the kidney in hypertension and in pregnancy. Specifically, he has been studying the role of the kidney in the development and maintenance of hypertension and the mechanisms that are responsible for renal and cardiovascular adaptations to normal pregnancy. His long term objective is to study complicated pregnancies such as diabetic and hypertensive pregnancies.

Biography Updated on 15 March 2007

Articles in Scholarly Journals [Incomplete List]

  1. Renal Interstitial Hydrostatic Pressure and Natriuretic Response to High Doses of Angiotensin II in Pregnant Rats
    American Journal of Hypertension, vol. 19, no. 3, pp. 300–305, 2006
  2. Natriuretic Response to Direct Renal Interstitial Volume Expansion (DRIVE) in Pregnant Rats
    American Journal of Hypertension, vol. 18, no. 6, pp. 851–857, 2005
  3. Role of RIHP and Renal Tubular Sodium Transporters in Volume Retention of Pregnant Rats
    American Journal of Hypertension, vol. 18, no. 10, pp. 1375–1383, 2005
  4. Cardiovascular and renal characteristics, and responses to acute volume expansion of a rat model of diabetic pregnancy
    Life Sciences, vol. 74, no. 23, pp. 2909–2918, 2004
  5. Effects of insulin on renal interstitial hydrostatic pressure and natriuretic response to volume expansion in diabetic rats
    AJP: Regulatory, Integrative and Comparative Physiology, vol. 286, no. 4, pp. 751R–755, 2004
  6. Pioglitazone Prevents Hypertension and Reduces Oxidative Stress in Diet-Induced Obesity
    Hypertension, vol. 43, no. 1, pp. 48–56, 2003
  7. Journal of Hypertension, vol. 20, no. 12, 2002
  8. Role of plasma renin activity and the renal nerves in the natriuresis of ?-NMMA infusion in rats
    Life Sciences, vol. 69, no. 10, pp. 1123–1131, 2001
  9. Role of gender on renal interstitial hydrostatic pressure and sodium excretion in rats
    American Journal of Hypertension, vol. 14, no. 9, pp. 893–896, 2001
  10. Blockade of cytochrome P-450 epoxygenase pathway attenuates the natriuresis of NG-monomethyl--arginine infusion in the spontaneously hypertensive rat1
    Journal of Laboratory and Clinical Medicine, vol. 129, no. 3, pp. 330–336, 1997
  11. The natriuresis produced by nitric oxide (NO) inhibition in the SHR is independent of changes in atrial natriuretic peptide (ANP).
    American Journal of Hypertension, vol. 9, no. 4, p. 126A, 1996
  12. Thymectomy Delays the Development of Hypertension in Okamoto Spontaneously Hypertensive
    Journal of Hypertension, vol. 5, no. 5, pp. 537–541, 1987