Fahumiya Samad, holder of a Ph.D. degree, is an Associate Professor at Torrey Pines Institute for Molecular Studies, Calif, USA. Dr. Samad received her Ph.D. degree in biology from the University of Wisconsin and completed a postdoctoral training at the Scripps Research Institute after which she joined the Department of Vascular Biology at Scripps as an Assistant Professor. Dr. Samad’s research focuses on the contribution of adipose tissue to the pathogenesis of obesity and related health consequences including insulin resistance, type 2 diabetes, fatty liver, and increased risk for cardiovascular disease and cancer. She was a pioneer in demonstrating an endocrine role for adipose tissue by demonstrating that a number of prothrombotic and inflammatory proteins are expressed in adipocytes/adipose tissues and elevated in obesity. She has worked and published extensively on prothrombotic (plasminogen activator inhibitor-1, tissue factor), inflammatory mediators, and related signaling pathways upregulated in the obese adipose tissue that may contribute to the pathogenesis of the metabolic syndrome. Her most recent research suggests that sphingolipids, including ceramide, sphingosine, and sphingosine 1 phosphate, are potential lipid signaling molecules that may link elevated levels of free fatty acids and inflammation due to obesity to insulin resistance/type 2 diabetes and cardiovascular risk. Dr. Samad has been funded by grants from the National Institutes of Health, the American Heart Association, and the Department of Defense's Congressionally Directed Medical Research Program. She reviews regularly for a number of international journals including the Journal of Clinical Investigation, PNAS, Journal of Biological Chemistry, Diabetes, Endocrinology, Circulation, American Journal of Physiology, and Blood. She has also reviewed grants for the American Heart Association and the NIH.
Biography Updated on 1 November 2010