Shimon Amir

Articles in Scholarly Journals [Incomplete List]

  1. Restricted access to food, but not sucrose, saccharine, or salt, synchronizes the expression of Period2 protein in the limbic forebrain
    Neuroscience, vol. 144, no. 2, pp. 402–411, 2007
  2. Differential regulation of the expression of Period2 protein in the limbic forebrain and dorsomedial hypothalamus by daily limited access to highly palatable food in food-deprived and free-fed rats
    Neuroscience, vol. 147, no. 2, pp. 277–285, 2007
  3. Pinealectomy does not affect diurnal PER2 expression in the rat limbic forebrain
    Neuroscience Letters, vol. 399, no. 1-2, pp. 147–150, 2006
  4. Thyroidectomy alters the daily pattern of expression of the clock protein, PER2, in the oval nucleus of the bed nucleus of the stria terminalis and central nucleus of the amygdala in rats
    Neuroscience Letters, vol. 407, no. 3, pp. 254–257, 2006
  5. Glucocorticoid rhythms control the rhythm of expression of the clock protein, Period2, in oval nucleus of the bed nucleus of the stria terminalis and central nucleus of the amygdala in rats
    Neuroscience, vol. 140, no. 3, pp. 753–757, 2006
  6. The expression of the clock protein PER2 in the limbic forebrain is modulated by the estrous cycle
    Proceedings of the National Academy of Sciences, vol. 103, no. 14, pp. 5591–5596, 2006
  7. Polymyxin B and Related Cyclic Peptides Facilitate Leanness and Reduce Fat Mass and Triglyceride Content in Ageing Rats: Potential Prototype Drugs Against Obesity
    International Journal of Peptide Research and Therapeutics, vol. 12, no. 2, pp. 121–129, 2006
  8. The central and basolateral nuclei of the amygdala exhibit opposite diurnal rhythms of expression of the clock protein Period2
    Proceedings of the National Academy of Sciences, vol. 102, no. 11, pp. 4180–4184, 2005
  9. Daily restricted feeding rescues a rhythm of period2 expression in the arrhythmic suprachiasmatic nucleus
    Neuroscience, vol. 132, no. 2, pp. 245–248, 2005
  10. Fos Expression in the Suprachiasmatic Nucleus During Photic Entrainment of Circadian Rhythms in Retinally Damaged Rats
    Journal of Molecular Neuroscience, vol. 22, no. 3, pp. 223–230, 2004
  11. A Circadian Rhythm in the Expression of PERIOD2 Protein Reveals a Novel SCN-Controlled Oscillator in the Oval Nucleus of the Bed Nucleus of the Stria Terminalis
    Journal of Neuroscience, vol. 24, no. 4, pp. 781–790, 2004
  12. Melanopsin in the Circadian Timing System
    Journal of Molecular Neuroscience, vol. 21, no. 1, pp. 73–90, 2003
  13. Expression Profiles of PER2 Immunoreactivity Within the Shell and Core Regions of the Rat Suprachiasmatic Nucleus: Lack of Effect of Photic Entrainment and Disruption by Constant Light
    Journal of Molecular Neuroscience, vol. 21, no. 2, pp. 133–148, 2003
  14. The eyes suppress a circadian rhythm of FOS expression in the suprachiasmatic nucleus in the absence of light
    Neuroscience, vol. 121, no. 2, pp. 253–257, 2003
  15. Prolactin and Oxytocin Interaction in the Paraventricular and Supraoptic Nuclei: Effects on Oxytocin mRNA and Nitric Oxide Synthase
    Journal of Neuroendocrinology, vol. 15, no. 7, pp. 687–696, 2003
  16. Effect of 192 IgG-Saporin on Circadian Activity Rhythms, Expression of P75 Neurotrophin Receptors, Calbindin-D28K, and Light-Induced Fos in the Suprachiasmatic Nucleus in Rats
    Experimental Neurology, vol. 176, no. 2, pp. 377–389, 2002
  17. Photic regulation of circadian rhythms and the expression of p75 neurotrophin receptor immunoreactivity in the suprachiasmatic nucleus in rats
    Brain Research, vol. 894, no. 2, pp. 301–306, 2001
  18. A novel, associative process modulating photic resetting of the circadian clock
    Neuroscience, vol. 104, no. 3, pp. 615–618, 2001
  19. Light suppresses Fos expression in the shell region of the suprachiasmatic nucleus at dusk and dawn: implications for photic entrainment of circadian rhythms
    Neuroscience, vol. 106, no. 2, pp. 249–254, 2001
  20. Enhanced Fos expression within the primary olfactory and limbic pathways induced by an aversive conditioned odor stimulus
    Neuroscience, vol. 98, no. 3, pp. 403–406, 2000
  21. Calbindin-D28k immunoreactivity in the suprachiasmatic nucleus and the circadian response to constant light in the rat
    Neuroscience, vol. 99, no. 3, pp. 397–401, 2000
  22. Time acts as a conditioned stimulus to control behavioral sensitization to amphetamine in rats
    Neuroscience, vol. 101, no. 1, pp. 1–3, 2000
  23. Circadian modulation of Fos responses to odor of the red fox, a rodent predator, in the rat olfactory system
    Brain Research, vol. 866, no. 1-2, pp. 262–267, 2000
  24. Expression profiles of JunB and c-Fos proteins in the rat circadian system
    Brain Research, vol. 870, no. 1-2, pp. 54–65, 2000
  25. Conditioned Stimulus Control in the Circadian System: Two Tales Tell One Story
    Journal of Biological Rhythms, vol. 15, no. 4, pp. 292–293, 2000
  26. Resetting the rat circadian clock by ultra-short light flashes
    Neuroscience Letters, vol. 261, no. 3, pp. 159–162, 1999
  27. In rats, odor-induced Fos in the olfactory pathways depends on the phase of the circadian clock
    Neuroscience Letters, vol. 272, no. 3, pp. 175–178, 1999
  28. Photic entrainment and induction of immediate-early genes within the rat circadian system
    Brain Research, vol. 821, no. 1, pp. 95–100, 1999
  29. Environmental Lighting Has a Selective Influence on Ethanol Intake in Rats
    Physiology & Behavior, vol. 66, no. 2, pp. 323–328, 1999
  30. Conditioned Fear Attenuates Light-Induced Suppression of Melatonin Release in Rats
    Physiology & Behavior, vol. 67, no. 4, pp. 623–626, 1999
  31. Changes in NADPH-d Staining in the Paraventricular and Supraoptic Nuclei During Pregnancy and Lactation in Rats: Role of Ovarian Steroids and Oxytocin
    Journal of Neuroendocrinology, vol. 11, no. 1, pp. 53–61, 1999
  32. The effectiveness of light on the circadian clock is linked to its emotional value
    Neuroscience, vol. 88, no. 2, pp. 339–345, 1999
  33. The intergeniculate leaflet does not mediate the disruptive effects of constant light on circadian rhythms in the rat
    Neuroscience, vol. 90, no. 3, pp. 1093–1101, 1999
  34. Conditioned and unconditioned aversive stimuli enhance light-induced Fos expression in the primary visual cortex
    Neuroscience, vol. 89, no. 2, pp. 323–327, 1999
  35. Olfactory stimulation enhances light-induced phase shifts in free-running activity rhythms and Fos expression in the suprachiasmatic nucleus
    Neuroscience, vol. 92, no. 4, pp. 1165–1170, 1999
  36. Glutamatergic antagonists do not attenuate light-induced Fos protein in rat intergeniculate leaflet
    Brain Research, vol. 810, no. 1-2, pp. 264–268, 1998
  37. Induction of Fos expression in the circadian system by unsignaled light is attenuated as a result of previous experience with signaled light: a role for Pavlovian conditioning
    Neuroscience, vol. 83, no. 3, pp. 657–661, 1998
  38. A role for serotonin in the circadian system revealed by the distribution of serotonin transporter and light-induced Fos immunoreactivity in the suprachiasmatic nucleus and intergeniculate leaflet
    Neuroscience, vol. 84, no. 4, pp. 1059–1073, 1998
  39. Conditioned fear suppresses light-induced resetting of the circadian clock
    Neuroscience, vol. 86, no. 2, pp. 345–351, 1998
  40. Correspondence
    Neuroscience, vol. 77, no. 3, pp. 623–627, 1997
  41. Lactation reduces Fos induction in the paraventricular and supraoptic nuclei of the hypothalamus after urethane administration in rats
    Brain Research, vol. 752, no. 1-2, pp. 319–323, 1997
  42. A blocker of nitric oxide synthase, NG-nitro-?-arginine methyl ester, attenuates light-induced Fos protein expression in rat suprachiasmatic nucleus
    Neuroscience Letters, vol. 224, no. 1, pp. 29–32, 1997
  43. Reproductive state changes NADPH-diaphorase staining in the paraventricular and supraoptic nuclei of female rats
    Brain Research, vol. 739, no. 1-2, pp. 339–342, 1996
  44. Resetting of the circadian clock by a conditioned stimulus
    Nature, vol. 379, no. 6565, Article ID 379542a0, 3 pages, 1996
  45. Fos expression in rat visual cortex induced by ocular input of ultraviolet light
    Brain Research, vol. 716, no. 1-2, pp. 213–218, 1996
  46. The nitric oxide synthase inhibitor NW-Nitro-L-Arginine methylester attenuates brain catalase activity in vitro
    Life Sciences, vol. 56, no. 16, pp. 1321–1324, 1995
  47. Distribution of NADPH-diaphorase staining and light-induced fos expression in the rat suprachiasmatic nucleus region supports a role for nitric oxide in the circadian system
    Neuroscience, vol. 69, no. 2, pp. 545–555, 1995
  48. Ultraviolet light entrains rodent suprachiasmatic nucleus pacemaker
    Neuroscience, vol. 69, no. 4, pp. 1005–1011, 1995
  49. Neonatal monosodium glutamate treatment prevents effects of constant light on circadian temperature rhythms of adult rats
    Brain Research, vol. 675, no. 1-2, pp. 135–142, 1995
  50. Induction of Fos protein in a model of closed head injury in rats
    Brain Research, vol. 690, no. 1, pp. 48–54, 1995
  51. Non-photic manipulations induce expression of Fos protein in the suprachiasmatic nucleus and intergeniculate leaflet in the rat
    Brain Research, vol. 690, no. 2, pp. 254–258, 1995
  52. Thermogenesis in brown adipose tissue is activated by electrical stimulation of the rat dorsal raphe nucleus
    Brain Research, vol. 650, no. 1, pp. 149–152, 1994
  53. Induction of Fos protein in the piriform cortex after brain injury in pentobarbital-anaesthetized rats: lack of effect of lactation
    Brain Research, vol. 652, no. 2, pp. 341–345, 1994
  54. N-methyl-D-aspartate receptor-mediated signaling in the supraoptic nucleus involves activation of a nitric oxide-dependent pathway
    Brain Research, vol. 645, no. 1-2, pp. 330–334, 1994
  55. Inhibition of nitric oxide synthase does not block the development of sensitization to the behavioral activating effects of amphetamine
    Brain Research, vol. 641, no. 1, pp. 141–144, 1994
  56. The effects of prostaglandin E2 injected into the paraventricular nucleus of the hypothalamus on brown adipose tissue thermogenesis in spontaneously hypertensive rats
    Brain Research, vol. 613, no. 2, pp. 285–287, 1993
  57. Blocking NMDA receptors or nitric oxide production disrupts light transmission to the suprachiasmatic nucleus
    Brain Research, vol. 586, no. 2, pp. 336–339, 1992
  58. NG-Monomethyl-l-arginine co-injection attenuates the thermogenic and hyperthermic effects of E2 prostaglandin microinjection into the anterior hypothalamic preoptic area in rats
    Brain Research, vol. 556, no. 1, pp. 157–160, 1991
  59. Activation of brown adipose tissue thermogenesis by chemical stimulation of the hypothalamic supraoptic nucleus
    Brain Research, vol. 563, no. 1-2, pp. 349–352, 1991
  60. An inhibitor of nitric oxide production, NGnitro-L-arginine-methyl ester, improves survival in anaphylactic shock
    European Journal of Pharmacology, vol. 203, no. 1, pp. 125–127, 1991
  61. Activation of brown adipose tissue thermogenesis by chemical stimulation of the posterior hypothalamus
    Brain Research, vol. 534, no. 1-2, pp. 303–308, 1990
  62. Intra-ventromedial hypothalamic injection of glutamate stimulates brown adipose tissue thermogenesis in the rat
    Brain Research, vol. 511, no. 2, pp. 341–344, 1990
  63. Injection of prostaglandin E2 into the anterior hypothalamic preoptic area activates brown adipose tissue thermogenesis in the rat
    Brain Research, vol. 528, no. 1, pp. 138–142, 1990
  64. Stimulation of the paraventricular nucleus with glutamate activates interscapular brown adipose tissue thermogenesis in rats
    Brain Research, vol. 508, no. 1, pp. 152–155, 1990
  65. Insulin co-injection suppresses the thermogenic response to glutamate microinjection into the VMH in rats
    Brain Research, vol. 527, no. 2, pp. 326–329, 1990
  66. Intra-ventromedial hypothalamic injection of insulin suppresses brown fat thermogenesis in the anaesthetized rat*1
    Brain Research, vol. 480, no. 1-2, pp. 340–343, 1989
  67. Glutamate injection into the suprachiasmatic nucleus stimulates brown fat thermogenesis in the rat
    Brain Research, vol. 498, no. 1, pp. 140–144, 1989
  68. Intra-hypothalamic injection of thyrotropin-releasing hormone suppresses brown fat thermogenesis in the anaesthetized rat
    Brain Research, vol. 478, no. 2, pp. 361–364, 1989
  69. Retinohypothalamic tract stimulation activates thermogenesis in brown adipose tissue in the rat
    Brain Research, vol. 503, no. 1, pp. 163–166, 1989
  70. Vanadate stimulates in vivo glucose uptake in brain and arrests food intake and body weight gain in rats
    Physiology & Behavior, vol. 45, no. 6, pp. 1113–1116, 1989
  71. Opposite effects of restraint on morphine analgesia and naloxone-induced jumping
    Pharmacology Biochemistry and Behavior, vol. 30, no. 4, pp. 905–910, 1988
  72. Thyrotropin-releasing hormone blocks neurally-mediated hyperglycemia through central action
    Peptides, vol. 9, no. 1, pp. 31–35, 1988
  73. Prevention of clonidine-stimulated hyperglycemia by thyrotropin-releasing hormone
    Peptides, vol. 9, no. 3, pp. 527–531, 1988
  74. Thyrotropin-releasing hormone (TRH) blocks glucagon-induced hyperglycemia in mice: dissociation of the antihyperglycemic and pituitary actions of TRH
    Brain Research, vol. 455, no. 1, pp. 201–203, 1988
  75. Immunological blockade of endogenous thyrotropin-releasing hormone impairs recovery from hyperglycemia in mice
    Brain Research, vol. 462, no. 1, pp. 160–162, 1988
  76. Aging blocks the thermoregulatory action of thyrotropin-releasing hormone in anaesthetized rats
    Brain Research, vol. 440, no. 1, pp. 181–184, 1988
  77. Apparent involvement of protein kinase C in the central glucoregulatory action of insulin
    Brain Research, vol. 450, no. 1-2, pp. 272–279, 1988
  78. TPA (12-O-tetradecanoylphorbol-13-acetate enhances the central hypoglycemic action of thyrotropin-releasing hormone in mice
    Brain Research, vol. 450, no. 1-2, pp. 369–372, 1988
  79. The use of post-binding agents in studying insulin action and its relation to experimental diabetes
    Biochemical Pharmacology, vol. 37, no. 10, pp. 1891–1896, 1988
  80. Thyrotropin-releasing hormone (TRH): Insulin-like action on glucoregulation
    Biochemical Pharmacology, vol. 37, no. 22, pp. 4245–4251, 1988
  81. Glucose transport
    Nature, vol. 336, no. 6199, Article ID 336528b0, 1 pages, 1988
  82. Vanadate ions: central nervous system action on glucoregulation
    Brain Research, vol. 419, no. 1-2, pp. 392–396, 1987
  83. Centrally mediated hypoglycemic effect of insulin: apparent involvement of specific insulin receptors
    Brain Research, vol. 418, no. 1, pp. 152–156, 1987
  84. Thyrotropin-releasing hormone potently reverses epinephrine-stimulated hyperglycemia in mice
    Brain Research, vol. 435, no. 1-2, pp. 112–122, 1987
  85. Central glucagon-induced hyperglycemia is mediated by combined activation of the adrenal medulla and sympathetic nerve endings
    Physiology & Behavior, vol. 37, no. 4, pp. 563–566, 1986
  86. Catalepsy Induced by Body Pinch: Relation to Stress-Induced Analgesia
    Annals of the New York Academy of Sciences, vol. 467, no. 1 Stress-Induce, pp. 226–237, 1986
  87. Polymyxin B is a potent inhibitor of insulin hypoglycemia in mice
    European Journal of Pharmacology, vol. 110, no. 2, pp. 283–285, 1985
  88. Systemic hypoglycemia following central injection of endotoxin in mice
    Brain Research, vol. 339, no. 2, pp. 382–385, 1985
  89. Beneficial effect of ?-endorphin-type peptides in anaphylactic shock
    Brain Research, vol. 329, no. 1-2, pp. 329–333, 1985
  90. Central thyrotropin-releasing hormone elicits systemic hypoglycemia in mice
    Brain Research, vol. 344, no. 2, pp. 387–391, 1985
  91. Naloxone improves, and morphine exacerbates, experimental shock induced by release of endogenous histamine by compound 48/80
    Brain Research, vol. 297, no. 1, pp. 187–190, 1984
  92. Beneficial effect of i.c.v. naloxone in anaphylactic shock is mediated through peripheral ß-adrenoceptive mechanisms
    Brain Research, vol. 290, no. 1, pp. 191–194, 1984
  93. Thyrotropin-releasing hormone (TRH) improves survival in anaphylactic shock: A central effect mediated by the sympatho-adrenomedullary ß-adrenoceptive system
    Brain Research, vol. 298, no. 2, pp. 219–224, 1984
  94. Anti-anaphylactic action in the mouse of thyrotropin-releasing hormone (TRH) is mediated through ß1-adrenoceptive effectors
    Neuroscience Letters, vol. 46, no. 2, pp. 127–130, 1984
  95. Opiate antagonists reverse the hypoactivity associated with systemic anaphylaxis in mice
    Pharmacology Biochemistry and Behavior, vol. 20, no. 3, pp. 483–485, 1984
  96. Role of endorphins in endotoxin-induced hyperglycaemia in mice
    Neuropharmacology, vol. 22, no. 9, pp. 1117–1119, 1983
  97. Morphine exacerbates anaphylactic shock in mice by stimulating central opiate receptors
    Neuroscience Letters, vol. 40, no. 2, pp. 169–174, 1983
  98. Proanaphylactic action of morphine is mediated through a central cholinergic mechanism
    European Journal of Pharmacology, vol. 95, no. 1-2, pp. 117–119, 1983
  99. Regulation of opiate receptors in mouse brain: arcuate nuclear lesion induces receptor up-regulation and supersensitivity to opiates
    Brain Research, vol. 262, no. 1, pp. 168–171, 1983
  100. Antianaphylactic effect of naloxone in mice is mediated by increased central sympathetic outflow to sympathetic nerve endings and adrenal medulla
    Brain Research, vol. 274, no. 1, pp. 180–183, 1983
  101. Opiate antagonists improve survival in anaphylactic shock
    European Journal of Pharmacology, vol. 80, no. 1, pp. 161–162, 1982
  102. Protective effect of neonatal monosodium glutamate-induced arcuate nucleus lesions in anaphylactic shock
    European Journal of Pharmacology, vol. 84, no. 3-4, pp. 237–238, 1982
  103. Involvement of endogenous opioids with forced swimming-induced immobility in mice
    Physiology & Behavior, vol. 28, no. 2, pp. 249–251, 1982
  104. Endogenous opioids interact in stress-induced hyperglycemia in mice
    Physiology & Behavior, vol. 28, no. 3, pp. 575–577, 1982
  105. A simple and adjustable restraining apparatus for mice
    Physiology & Behavior, vol. 26, no. 2, pp. 335–336, 1981
  106. Behavioral response of the genetically obese (ob/ob) mouse to heat stress: Effects of naloxone and prior exposure to immobilization stress
    Physiology & Behavior, vol. 27, no. 2, pp. 249–253, 1981
  107. Effects of ACTH on pain responsiveness in mice: Interaction with morphine
    Neuropharmacology, vol. 20, no. 10, pp. 959–962, 1981
  108. Chronic lithium administration alters the interaction between opiate antagonists and opiate receptors in vivo
    Neuropharmacology, vol. 20, no. 6, pp. 587–591, 1981
  109. Body pinch induces long lasting cataleptic like immobility in mice: Behavioral characterization and the effect of naloxone
    Life Sciences, vol. 28, no. 10, pp. 1189–1194, 1981
  110. Opiate receptors may mediate the suppressive but not the excitatory action of ACTH on motor activity in rats
    European Journal of Pharmacology, vol. 66, no. 4, pp. 307–313, 1980
  111. The role of endorphins in stress: Evidence and speculations
    Neuroscience & Biobehavioral Reviews, vol. 4, no. 1, pp. 77–86, 1980
  112. Enhanced analgesic effects of stress following chronic administration of naltrexone in rats
    European Journal of Pharmacology, vol. 59, no. 1-2, pp. 137–140, 1979
  113. The effect of acute and chronic naloxone administration on motor activation in the rat
    Neuropharmacology, vol. 18, no. 2, pp. 171–173, 1979
  114. Chronic naltrexone administration reverses the suppressive effect of crowding on body weight gain in rats
    Neuropharmacology, vol. 18, no. 11, pp. 905–907, 1979
  115. The pituitary gland mediates acute and chronic pain responsiveness in stressed and non-stressed rats
    Life Sciences, vol. 24, no. 5, pp. 439–448, 1979
  116. Increased amphetamine potency following chronic naltrexone administration in rats
    Life Sciences, vol. 25, no. 16, pp. 1407–1412, 1979
  117. Endogenous opioid ligands may mediate stress-induced changes in the affective properties of pain related behavior in rats
    Life Sciences, vol. 23, no. 11, pp. 1143–1151, 1978
  118. Ventral hypothalamic lesions block the consumption of morphine in rats
    Life Sciences, vol. 13, no. 7, pp. 805–816, 1973