Allal Ouhtit

Allal Ouhtit is an Assistant Professor of Pathology. He received his M.P.H. degree in neuroscience from the University of Lyon in France. His Ph.D. degree in biochemistry was awarded in early 1995 from the University of Lyon. He completed his postdoctoral work at the International Agency for research on Cancer (World Health Organization) and joined MD Anderson Cancer Center in 1997. In 2001, Dr. Ouhtit moved to the Queens University of Belfast in Northern Ireland as an Assistant Professor. In 2005, he was appointed as an Assistant Professor at LSU Health Sciences Center. Dr. Ouhtit’s research is centered on the mechanisms of CD44 signaling in invasion/metastasis of melanoma, breast, and prostate cancers. His laboratory is also carrying out translational/basic research on Emphysema and skin diseases including skin cancers. He has been a Member of the Stanley S. Scott Cancer Center since 2005.

Biography Updated on 15 March 2007

Articles in Scholarly Journals [Incomplete List]

  1. Survivin is not only a death encounter but also a survival protein for invading tumor cells
    Frontiers in Bioscience, vol. 12, no. 1, p. 1260, 2007
  2. Hydrogen peroxide acts as relaxing factor in human vascular smooth muscle cells independent of map-kinase and nitric oxide
    Frontiers in Bioscience, vol. 11, no. 1, p. 2526, 2006
  3. The effect of ultra violet B (TL-01) phototherapy on epidermal expression of p53 protein in psoriatic plaques
    Photodermatology, Photoimmunology and Photomedicine, vol. 22, no. 1, pp. 12–17, 2006
  4. Cortactin underpins CD44-promoted invasion and adhesion of breast cancer cells to bone marrow endothelial cells
    Oncogene, vol. 25, no. 45, Article ID 1209628, 12 pages, 2006
  5. Towards a Better Understanding of the Molecular Mechanisms Involved in Sunlight-Induced Melanoma
    Journal of Biomedicine and Biotechnology, vol. 2005, no. 1, pp. 57–61, 2005
  6. Dysfunction of p53 in photocarcinogenesis
    Frontiers in Bioscience, vol. 8, no. 1-3, p. s715, 2003
  7. A Model for UV-Induction of Skin Cancer
    Journal of Biomedicine and Biotechnology, vol. 1, no. 1, pp. 5–6, 2001
  8. UV irradiation augments lymphoid malignancies in mice with one functional copy of wild-type p53
    Proceedings of the National Academy of Sciences, vol. 98, no. 17, pp. 9790–9795, 2001
  9. UVB-induced experimental carcinogenesis: dysregulation of apoptosis and p53 signalling pathway
    Redox Report, vol. 5, no. 2, pp. 128–129, 2000
  10. Persistence of p53 mutations and resistance of keratinocytes to apoptosis are associated with the increased susceptibility of mice lacking the XPC gene to UV carcinogenesis
    Oncogene, vol. 18, no. 51, pp. 7395–7398, 1999
  11. Fas Ligand: A Sensor for DNA Damage Critical in Skin Cancer Etiology
    Science, vol. 285, no. 5429, pp. 898–900, 1999
  12. UV-radiation-specific p53 mutation frequency in normal skin as a predictor of risk of basal cell carcinoma
    Journal of the National Cancer Institute, vol. 90, no. 7, pp. 523–531, 1998
  13. Visualization of gene expression of prolactin receptors (PRL-R) by in situ hybridization, in Typhlonectes compressicaudus, a gymnophionan amphibian
    Life Sciences, vol. 61, no. 19, pp. 1915–1928, 1997
  14. Ultrastructural expression of prolactin receptor in rat liver
    Biology of the Cell, vol. 82, no. 2-3, pp. 169–176, 1994
  15. Visualization of gene expression of short and long forms of prolactin receptor in the rat
    Endocrinology, vol. 133, no. 1, pp. 135–144, 1993
  16. Visualization of gene expression of short and long forms of prolactin receptor in rat reproductive tissues
    Biology of Reproduction, vol. 49, no. 3, pp. 528–536, 1993