﻿<?xml version="1.0" encoding="utf-8"?><rss version="2.0"><channel><title>Anesthesiology Research and Practice</title><link>http://www.hindawi.com</link><description>The latest articles from Hindawi Publishing Corporation</description><copyright>&amp;#169; 2008, Hindawi Publishing Corporation. All rights reserved.</copyright><item><title>Physiologic Responses to Infrarenal Aortic Cross-Clamping during Laparoscopic or Conventional Vascular Surgery in Experimental Animal Model: Comparative Study</title><link>http://www.hindawi.com/GetArticle.aspx?doi=10.1155/2008/581948</link><description>The aim of this study was to compare the hemodynamic and ventilatory effects of prolonged infrarenal aortic cross-clamping in pigs undergoing either laparotomy or laparoscopy.
18 pigs were used for this study. 
Infrarenal aortic crossclamping was performed for 60 minutes in groups
 I (laparotomy, n=6) and II (laparoscopy, n=6). Group III (laparoscopy, n=6) underwent a 120-minute long pneumoperitoneum in absence of aortic clamping (sham group). 
 Ventilatory and hemodynamic parameters and renal function were serially determined in all groups. 
A significant decrease in pH and significant increase in PaCO2 were observed in group II, whereas no changes in these parameters were seen in group I and III. All variables returned to values similar to baseline in groups I and II 60 minutes after declamping. A significant increase in renal resistive index was evidenced during laparoscopy, with significantly higher values seen in Group II. 
Thus a synergic effect of pneumoperitoneum and aortic cross-clamping was seen in this study. These two factors together cause decreased renal perfusion and acidosis, thus negatively affecting the patient&amp;#39;s general state during this type of surgery.</description><Author>Mar&amp;#237;a F. Mart&amp;#237;n-Cancho, Ver&amp;#243;nica Cris&amp;#243;stomo, Federico Soria, Carmen Calles, Francisco M. S&amp;#225;nchez-Margallo, Idoia D&amp;#237;az-G&amp;#252;emes, and Jes&amp;#250;s Us&amp;#243;n-Gargallo</Author><copyright>&amp;#169; 2008, Hindawi Publishing Corporation. All rights reserved.</copyright></item><item><title>Acute Inflammatory Demyelinating Polyneuropathy and a Unilateral 
Babinski/Plantar Reflex</title><link>http://www.hindawi.com/GetArticle.aspx?doi=10.1155/2008/134958</link><description>Acquired acute demyelinating peripheral 
polyneuropathy (AADP) is a general classification of pathologies 
that could affect secondary the peripheral nervous system. They 
are characterized by an autoimmune process directed towards 
myelin. Clinically they are characterized by progressive weakness 
and mild sensory changes. Acute inflammatory demyelinating 
polyneuropathy often is referred to as Guillain-Barr&amp;#233; syndrome 
(GBS). GBS is the major cause of acute nontraumatic paralysis in 
healthy people and it is caused by autoimmune response to viral 
agents (influenza, coxsackie, Epstein-Barr virus, or 
cytomegalovirus) or bacterial infective organisms (Campylobacter 
jejuni, Mycoplasma pneumoniae). A detailed history, with symptoms 
of progressive usually bilateral weakness, hyporeflexia, with a 
typical demyelinating EMG pattern supports the diagnosis. 
Progressive affection of respiratory muscles and autonomic 
instability coupled with a protracted and unpredictable recovery 
normally results in the need for ICU management. We present a case 
report of a patient with a typical GBS presentation but with a 
unilateral upgoing plantar reflex (Babinski sign). A unifying 
diagnosis was made and based on a literature search in Pubmed 
appears to be the first described case of its kind. 
</description><Author>Davide Cattano, Brian O&amp;#39;connor, Ra&amp;#39;ad Shakir, Francesco Giunta, and Mark Palazzo</Author><copyright>&amp;#169; 2008, Hindawi Publishing Corporation. All rights reserved.</copyright></item></channel></rss>