p53: The Attractive Tumor Suppressor in the Cancer Research Field

<table>Upon DNA damage, activated form of ATM phosphorylates IKK-<i>α</i> and promotes nuclear accumulation of IKK-<i>α</i>. IKK-<i>α</i> enhances transactivation as well as proapoptotic function of p73 in a p53-independent manner.</table>

BioMed Research International

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Figure 4

Figure 4: p53: The Attractive Tumor Suppressor in the Cancer Research Field 