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Journal of Biomedicine and Biotechnology
Volume 2012 (2012), Article ID 141806, 6 pages
So Many Plasminogen Receptors: Why?
Department of Molecular Cardiology, Joseph J. Jacobs Center for Thrombosis and Vascular Biology, Cleveland Clinic, 9500 Euclid Avenue, NB50, Cleveland, OH 44195, USA
Received 2 April 2012; Accepted 7 June 2012
Academic Editor: Lindsey A. Miles
Copyright © 2012 Edward F. Plow et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Citations to this Article [14 citations]
The following is the list of published articles that have cited the current article.
- Hanna M. Järvinen, Liisa Laakkonen, Johanna Haiko, Tiira Johansson, Katri Juuti, Marjo Suomalainen, Carmen Buchrieser, Nisse Kalkkinen, and Timo K. Korhonen, “ Human single-chain urokinase is activated by the omptins PgtE of Salmonella enterica and Pla of Yersinia pestis despite mutations of active site residues ,” Molecular Microbiology, 2013.
- Jane Hoover-Plow, and Menggui Huang, “Lipoprotein(a) metabolism: Potential sites for therapeutic targets,” Metabolism, vol. 62, no. 4, pp. 479–491, 2013.
- Laurent Plawinski, and Eduardo Anglés-Cano, “Fibrinolytic microvesicles,” Sang Thrombose Vaisseaux, vol. 25, no. 2, pp. 100–110, 2013.
- Lindseya. Miles, and Robertj. Parmer, “Plasminogen receptors: The first quarter century,” Seminars in Thrombosis and Hemostasis, vol. 39, no. 4, pp. 329–337, 2013.
- Rocco Romagnuolo, Santica M. Marcovina, and Michael B. Boffa, “Inhibition of plasminogen activation by apo(a): role of carboxyl-terminal lysines and identification of inhibitory domains in apo(a),” Journal of Lipid Research, vol. 55, no. 4, pp. 625–634, 2014.
- Hunter B. Moore, Ernest E. Moore, Eduardo Gonzalez, Michael P. Chapman, Theresa L. Chin, Christopher C. Silliman, Anirban Banerjee, and Angela Sauaia, “Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: The spectrum of postinjury fibrinolysis and relevance to antifibrinolytic ther,” Journal Of Trauma And Acute Care Surgery, vol. 77, no. 6, pp. 811–817, 2014.
- Miroslava Didiasova, Lukasz Wujak, Malgorzata Wygrecka, and Dariusz Zakrzewicz, “From Plasminogen to Plasmin: Role of Plasminogen Receptors in Human Cancer,” International Journal Of Molecular Sciences, vol. 15, no. 11, pp. 21229–21252, 2014.
- Reiko Ikeda, Tomoe Ichikawa, Yusuke Miyazaki, Nanaho Shimizu, Tomomi Ryoke, Kunihiko Haru, Takashi Sugita, and Masako Takashima, “ Detection and characterization of plasminogen receptors on clinical isolates of Trichosporon asahii ,” FEMS Yeast Research, 2014.
- Reiko Ikeda, and Tomoe Ichikawa, “ Interaction of surface molecules on Cryptococcus neoformans with plasminogen ,” FEMS Yeast Research, 2014.
- Claire S. Whyte, Frauke Swieringa, Tom G. Mastenbroek, Ausra S. Lionikiene, Marcus D. Lance, Paola E. J. van der Meijden, Johan W. M. Heemskerk, and Nicola J. Mutch, “Plasminogen associates with phosphatidylserine-exposing platelets and contributes to thrombus lysis under flow,” Blood, vol. 125, no. 16, pp. 2568–2578, 2015.
- Hunter B. Moore, Ernest E. Moore, Eduardo Gonzalez, Kirk C. Hansen, Monika Dzieciatkowska, Michael P. Chapman, Angela Sauaia, Bernadette West, Anirban Banerjee, and Christopher C. Silliman, “Hemolysis Exacerbates Hyperfibrinolysis, Whereas Platelolysis Shuts Down Fibrinolysis: Evolving Concepts Of The Spectrum Of Fibrinolysis In Response T,” Shock, vol. 43, no. 1, pp. 39–46, 2015.
- A. Laurenzana, G. Fibbi, F. Margheri, A. Biagioni, C. Luciani, M. Del Rosso, and A. Chilla, “Endothelial Progenitor Cells in Sprouting Angiogenesis: Proteases Pave the Way,” Current Molecular Medicine, vol. 15, no. 7, pp. 606–620, 2015.
- V. G. Trusca, E. V. Fuior, and A. V. Gafencu, “Beyond Lipoprotein Receptors: Learning from Receptor Knockouts Mouse Models about New Targets for Reduction of the Atherosclerotic Plaque,” Current Molecular Medicine, vol. 15, no. 10, pp. 905–931, 2015.
- Nissim Silanikove, “Transcellular route as the most probable explanation for the presence of plasminogen in mammal's milk,” Journal of Theoretical Biology, 2016.