MicroRNAs in Kidney Fibrosis and Diabetic Nephropathy: Roles on EMT and EndMT

<table>Biochemical changes during EndMT program. The EndMT program causes decreased expression of endothelial markers VE-cadherin, CD31, cytokeratins, and type 4 collagen and a gain of mesenchymal markers FSP-1, <i>α</i>SMA, N-cadherin, vimentin, fibronectin, type I and type III collagen, and MMP-2 and MMP-9. FSP-1: fibroblast-specific protein-1; <i>α</i>-SMA: <i>α</i>-smooth muscle actin; and MMP: matrix metalloproteinase.</table>

BioMed Research International

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Figure 3

Figure 3: MicroRNAs in Kidney Fibrosis and Diabetic Nephropathy: Roles on EMT and EndMT 