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BioMed Research International
Volume 2013 (2013), Article ID 463168, 4 pages
Highly Ordered Architecture of MicroRNA Cluster
1Department of Geriatric Cardiology, Beijing Military General Hospital, Beijing 100700, China
2The 14th Beijing High School, Beijing, 100020, China
3School of Computer Science and Engineering, Hebei University of Technology, Tianjin 300401, China
Received 18 July 2013; Accepted 3 September 2013
Academic Editor: Qinghua Cui
Copyright © 2013 Bing Shi et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
- S. B. Carroll, “Homeotic genes and the evolution of arthropods and chordates,” Nature, vol. 376, no. 6540, pp. 479–485, 1995.
- D. P. Bartel, “MicroRNAs: genomics, biogenesis, mechanism, and function,” Cell, vol. 116, no. 2, pp. 281–297, 2004.
- M. Sawera, J. Gorodkin, S. Cirera, and M. Fredholm, “Mapping and expression studies of the mir17-92 cluster on pig Chromosome 11,” Mammalian Genome, vol. 16, no. 8, pp. 594–598, 2005.
- M. Lu, Q. Zhang, M. Deng et al., “An analysis of human microRNA and disease associations,” PLoS ONE, vol. 3, no. 10, Article ID e3420, 2008.
- D. Wang, J. Wang, M. Lu, F. Song, and Q. Cui, “Inferring the human microRNA functional similarity and functional network based on microRNA-associated diseases,” Bioinformatics, vol. 26, no. 13, pp. 1644–1650, 2010.
- Y. Hayashita, H. Osada, Y. Tatematsu et al., “A polycistronic MicroRNA cluster, miR-17-92, is overexpressed in human lung cancers and enhances cell proliferation,” Cancer Research, vol. 65, no. 21, pp. 9628–9632, 2005.
- S. Willimott and S. D. Wagner, “Stromal cells and CD40 ligand (CD154) alter the miRNome and induce miRNA clusters including, miR-125b/miR-99a/let-7c and miR-17-92 in chronic lymphocytic leukaemia,” Leukemia, vol. 26, pp. 1113–1116, 2012.
- K. Conkrite, M. Sundby, S. Mukai et al., “Mir-17~92 cooperates with RB pathway mutations to promote retinoblastoma,” Genes and Development, vol. 25, no. 16, pp. 1734–1745, 2011.
- M. Valladares-Ayerbes, M. Blanco, M. Haz et al., “Prognostic impact of disseminated tumor cells and microRNA-17-92 cluster deregulation in gastrointestinal cancer,” International Journal of Oncology, vol. 39, no. 5, pp. 1253–1264, 2011.
- L. Chen, C. Li, R. Zhang et al., “MiR-17-92 cluster microRNAs confers tumorigenicity in multiple myeloma,” Cancer Letters, vol. 309, no. 1, pp. 62–70, 2011.
- T.-F. F. Chow, M. Mankaruos, A. Scorilas et al., “The miR-17-92 cluster is over expressed in and has an oncogenic effect on renal cell carcinoma,” Journal of Urology, vol. 183, no. 2, pp. 743–751, 2010.
- P. Mu, Y.-C. Han, D. Betel et al., “Genetic dissection of the miR-17-92 cluster of microRNAs in Myc-induced B-cell lymphomas,” Genes and Development, vol. 23, no. 24, pp. 2806–2811, 2009.
- P. A. Northcott, A. Fernandez-L, J. P. Hagan et al., “The miR-17/92 polycistron is up-regulated in sonic hedgehog-driven medulloblastomas and induced by N-myc in sonic hedgehog-treated cerebellar neural precursors,” Cancer Research, vol. 69, no. 8, pp. 3249–3255, 2009.
- T. Uziel, F. V. Karginov, S. Xie et al., “The miR-17-92 cluster collaborates with the Sonic Hedgehog pathway in medulloblastoma,” Proceedings of the National Academy of Sciences of the United States of America, vol. 106, no. 8, pp. 2812–2817, 2009.
- S. Nagel, L. Venturini, G. K. Przybylski et al., “Activation of miR-17-92 by NK-like homeodomain proteins suppresses apoptosis via reduction of E2F1 in T-cell acute lymphoblastic leukemia,” Leukemia and Lymphoma, vol. 50, no. 1, pp. 101–108, 2009.
- S. Takakura, N. Mitsutake, M. Nakashima et al., “Oncogenic role of miR-17-92 cluster in anaplastic thyroid cancer cells,” Cancer Science, vol. 99, no. 6, pp. 1147–1154, 2008.
- A. Rinaldi, G. Poretti, I. Kwee et al., “Concomitant MYC and microRNA cluster miR-17-92 (C13orf25) amplification in human mantle cell lymphoma,” Leukemia and Lymphoma, vol. 48, no. 2, pp. 410–412, 2007.
- Y. Liang, D. Ridzon, L. Wong, and C. Chen, “Characterization of microRNA expression profiles in normal human tissues,” BMC Genomics, vol. 8, article 166, 2007.
- D. Bell, A. Berchuck, M. Birrer, et al., “Integrated genomic analyses of ovarian carcinoma,” Nature, vol. 474, no. 7353, pp. 609–615, 2011.
- A. Keller, P. Leidinger, A. Bauer et al., “Toward the blood-borne miRNome of human diseases,” Nature Methods, vol. 8, no. 10, pp. 841–843, 2011.
- V. Olive, M. J. Bennett, J. C. Walker et al., “miR-19 is a key oncogenic component of mir-17-92,” Genes and Development, vol. 23, no. 24, pp. 2839–2849, 2009.
- A. Tsuchida, S. Ohno, W. Wu et al., “miR-92 is a key oncogenic component of the miR-17-92 cluster in colon cancer,” Cancer Science, vol. 102, no. 12, pp. 2264–2271, 2011.
- S. G. Chaulk, G. L. Thede, O. A. Kent et al., “Role of pri-miRNA tertiary structure in miR-17~92 miRNA biogenesis,” RNA Biology, vol. 8, no. 6, 2011.