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BioMed Research International
Volume 2013 (2013), Article ID 474963, 6 pages
Association between Inflammatory Marker, Environmental Lead Exposure, and Glutathione S-Transferase Gene
1Department of Medicine, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok 10400, Thailand
2Division of Clinical Pharmacology and Toxicology, Department of Medicine, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok 10400, Thailand
3Office of Research Academic and Innovation, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok 10400, Thailand
4Health Office, Electricity Generating Authority of Thailand, Nonthaburi 11130, Thailand
Received 2 November 2012; Accepted 12 December 2012
Academic Editor: Marija Mostarica-Stojković
Copyright © 2013 Jintana Sirivarasai et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
- H. Gurer-Orhan, H. U. Sabir, and H. Ozgunes, “Correlation between clinical indicators of lead poisoning and oxidative stress parameters in controls and lead-exposed workers,” Toxicology, vol. 195, no. 2-3, pp. 147–154, 2004.
- M. Pande and S. J. S. Flora, “Lead induced oxidative damage and its response to combined administration of α-lipoic acid and succimers in rats,” Toxicology, vol. 177, no. 2-3, pp. 187–196, 2002.
- J. H. Kim, K. H. Lee, D. H. Yoo, et al., “GSTM1 and TNF-α gene polymorphism and relations between blood lead and inflammatory markers in a non-occupational population,” Mutation Research, vol. 629, no. 1, pp. 32–39, 2007.
- D. A. Khan, S. Qayyum, S. Saleem, and F. A. Khan, “Lead-induced oxidative stress adversely affects health of the occupational workers,” Toxicology and Industrial Health, vol. 24, no. 9, pp. 611–618, 2008.
- R. S. Dwivedi, “Lead exposure alters the drug metabolic activity and the homeostasis of essential metal ions in the lenticular system of the rat,” Environmental Pollution, vol. 94, no. 1, pp. 61–66, 1996.
- D. A. Daggett, T. D. Oberley, S. A. Nelson, L. S. Wright, S. E. Kornguth, and F. L. Siegel, “Effects of lead on rat kidney and liver: GST expression and oxidative stress,” Toxicology, vol. 128, no. 3, pp. 191–206, 1998.
- N. Songdej, P. C. Winters, M. J. McCabe, and E. V. Wijngaarden, “A population-based assessment of blood lead levels in relation to inflammation,” Environmental Research, vol. 110, no. 3, pp. 272–277, 2010.
- P. Sritara, S. Cheepudomwit, N. Chapman et al., “Twelve-year changes in vascular risk factors and their associations with mortality in a cohort of 3499 Thais: the Electricity Generating Authority of Thailand study,” International Journal of Epidemiology, vol. 32, no. 3, pp. 461–468, 2003.
- K. S. Subramanian and J. C. Meranger, “A rapid electrothermal atomic absorption spectrophotometric method for cadmium and lead in human whole blood,” Clinical Chemistry, vol. 27, no. 11, pp. 1866–1871, 1981.
- R. Dominici, P. Luraschi, and C. Franzini, “Measurement of C-reactive protein: two high sensitivity methods compared,” Journal of Clinical Laboratory Analysis, vol. 18, no. 5, pp. 280–284, 2004.
- S. A. Miller, D. D. Dykes, and H. F. Polesky, “A simple salting out procedure for extracting DNA from human nucleated cells,” Nucleic Acids Research, vol. 16, no. 3, article 1215, 1988.
- B. R. Packer, M. Yeager, B. Stats, et al., “SNP 500 cancer: a public resource for sequence validation and assay development for genetic variation in candidate genes,” Nucleic Acids Research, vol. 32, pp. D528–D534, 2004.
- I. Iavicoli, G. Carelli, E. J. Stanek, N. Castellino, and E. J. Calabrese, “Below background levels of blood lead impact cytokine levels in male and female mice,” Toxicology and Applied Pharmacology, vol. 210, no. 1-2, pp. 94–99, 2006.
- G. Falq, A. Zeghnoun, M. Pascal et al., “Blood lead levels in the adult population living in France the French Nutrition and Health Survey (ENNS 2006-2007),” Environment International, vol. 37, no. 3, pp. 565–571, 2011.
- V. Wietlisbach, M. Rickenbach, M. Berode, and M. Guillemin, “Time trend and determinants of blood lead levels in a Swiss population over a transition period (1984–1993) from leaded to unleaded gasoline use,” Environmental Research, vol. 68, no. 2, pp. 82–90, 1995.
- M. Galazyn-Sidorczuk, M. M. Brzóska, and J. Moniuszko-Jakoniuk, “Estimation of Polish cigarettes contamination with cadmium and lead, and exposure to these metals via smoking,” Environmental Monitoring and Assessment, vol. 137, no. 1–3, pp. 481–493, 2008.
- D. Kim and D. A. Lawrence, “Immunotoxic effects of inorganic lead on host resistance of mice with different circling behavior preferences,” Brain, Behavior, and Immunity, vol. 14, no. 4, pp. 305–317, 2000.
- K. P. Mishra, V. K. Singh, R. Rani et al., “Effect of lead exposure on the immune response of some occupationally exposed individuals,” Toxicology, vol. 188, no. 2-3, pp. 251–259, 2003.
- J. I. Anetor and F. A. Adeniyi, “Decreased immune status in Nigerian workers occupationally exposed to lead,” African journal of medicine and medical sciences, vol. 27, no. 3-4, pp. 169–172, 1998.
- N. D. Vaziri and M. Khan, “Interplay of reactive oxygen species and nitric oxide in the pathogenesis of experimental lead-induced hypertension,” Clinical and Experimental Pharmacology and Physiology, vol. 34, no. 9, pp. 920–925, 2007.
- H. D. Intengan and E. L. Schiffrin, “Vascular remodeling in hypertension: roles of apoptosis, inflammation, and fibrosis,” Hypertension, vol. 38, no. 3, pp. 581–587, 2001.
- X. Guo, S. J. O'Brien, Y. Zeng, G. W. Nelson, and C. A. Winkler, “GSTM1 and GSTT1 gene deletions and the risk for nasopharyngeal carcinoma in Han Chinese,” Cancer Epidemiology Biomarkers and Prevention, vol. 17, no. 7, pp. 1760–1763, 2008.
- N. Khansakorn, P. Sritara, J. Sirivarasai, et al., “Impact of GSTM1, GSTT1, GSTP1 polymorphism and environmental lead exposure on oxidative stress biomarkers,” Scientific Research and Essays, vol. 6, no. 31, pp. 6540–6547, 2011.
- D. D. Kwon, J. W. Lee, D. Y. Han et al., “Relationship between the glutathione-S-transferase P1, M1, and T1 genotypes and prostate cancer risk in Korean subjects,” Korean Journal of Urology, vol. 52, no. 4, pp. 247–252, 2011.
- J. C. W. Mak, S. P. Ho, H. C. M. Leung et al., “Relationship between glutathione S-transferase gene polymorphisms and enzyme activity in Hong Kong Chinese asthmatics,” Clinical and Experimental Allergy, vol. 37, no. 8, pp. 1150–1157, 2007.
- C. Gundacker, K. J. Wittmann, M. Kukuckova, G. Komarnicki, I. Hikkel, and M. Gencik, “Genetic background of lead and mercury metabolism in a group of medical students in Austria,” Environmental Research, vol. 109, no. 6, pp. 786–796, 2009.
- J. Bernier, P. Brousseau, K. Krzystyniak, H. Tryphonas, and M. Fournier, “Immunotoxicity of heavy metals in relation to Great Lakes,” Environmental Health Perspectives, vol. 103, Supplement 9, pp. 23–34, 1995.
- Y. Heo, W. T. Lee, and D. A. Lawrence, “Differential effects of lead and cAMP on development and activities of Th1- and Th2-lymphocytes,” Toxicological Sciences, vol. 43, no. 2, pp. 172–185, 1998.
- F. G. Hage and A. J. Szalai, “C-reactive protein gene polymorphisms, C-reactive protein blood levels, and cardiovascular disease risk,” Journal of the American College of Cardiology, vol. 50, no. 12, pp. 1115–1122, 2007.
- G. M. Fuller and Z. Zhang, “Transcriptional control mechanism of fibrinogen gene expression,” Annals of the New York Academy of Sciences, vol. 936, pp. 469–479, 2001.