Erratum to “Modulation of Vasodilator Response via the Nitric Oxide Pathway after Acute Methyl Mercury Chloride Exposure in Rats”

<table>Effect of methyl mercury chloride treatment on the vascular relaxation response to acetylcholine, isoproterenol, and sodium nitroprusside. Concentration-response curves to (a) acetylcholine (ACh), (b) isoproterenol (IP), and (c) sodium nitroprusside (SNP) in the aortas of rats untreated, treated with methyl mercury chloride <svg height="15.0875" id="M1" style="vertical-align:-2.3205pt" version="1.1" viewbox="0 0 56.162498 15.0875" width="56.162498" xmlns="http://www.w3.org/2000/svg" xmlns:xlink="http://www.w3.org/1999/xlink">
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</svg> precontracted with PE. The results (mean ± SEM) are expressed as percentage of the response to PE. <svg height="10.3875" id="M2" style="vertical-align:-0.1638pt" version="1.1" viewbox="0 0 5.9250002 10.3875" width="5.9250002" xmlns="http://www.w3.org/2000/svg" xmlns:xlink="http://www.w3.org/1999/xlink">
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</svg>-test, <svg height="13.95" id="M3" style="vertical-align:-0.1638pt" version="1.1" viewbox="0 0 66.324997 13.95" width="66.324997" xmlns="http://www.w3.org/2000/svg" xmlns:xlink="http://www.w3.org/1999/xlink">
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</svg>.</table>

BioMed Research International

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Figure 2

Figure 2: Erratum to “Modulation of Vasodilator Response via the Nitric Oxide Pathway after Acute Methyl Mercury Chloride Exposure in Rats” 