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Autophagy in Development, Cell Differentiation, and Homeodynamics: From Molecular Mechanisms to Diseases and Pathophysiology

Call for Papers

Autophagy is an evolutionarily conserved process and plays an essential role in both cellular and whole organism homeodynamics. During autophagy, cytoplasmic components are sequestered into double-membrane vesicles called autophagosomes, which then fuse with lysosomes and their content is degraded. Autophagy is capable of responding to environmental and hormonal cues, to regulate cellular processes necessary for proper differentiation and development, and to control cellular homeodynamics. Failure of autophagy to perform these functions underlies the pathogenesis of several diseases.

We invite authors to submit original research articles as well as review articles that will stimulate the continuing efforts to understand the molecular mechanisms underlying the role of autophagy in development, cell differentiation, and homeodynamics during health and disease. Potential topics include, but are not limited to:

  • Autophagy during development in model organisms
  • Autophagy in cell differentiation
  • Autophagy and tissue homeostasis
  • Autophagy and proteostasis
  • Autophagy and ageing
  • Regulation of autophagy by hormonal, nutritional, and heterotypic signals

Before submission authors should carefully read over the journal’s Author Guidelines, which are located at http://www.hindawi.com/journals/bmri/guidelines/. Prospective authors should submit an electronic copy of their complete manuscript through the journal Manuscript Tracking System at http://mts.hindawi.com/submit/journals/bmri/cell.biology/autod/ according to the following timetable:

Manuscript DueFriday, 4 April 2014
First Round of ReviewsFriday, 27 June 2014
Publication DateFriday, 22 August 2014

Lead Guest Editor

  • Ioannis P. Nezis, School of Life Sciences, University of Warwick, Coventry, CV4 7AL, UK

Guest Editors

  • Maria Vaccaro, Institute for Biochemistry and Molecular Medicine(CONICET), School of Pharmacy and Biochemistry, University of Buenos Aires, 1113 Buenos Aires, Argentina
  • Rod Devenish, Department of Biochemistry and Molecular Biology, Monash University, Clayton Campus, Melbourne, VIC, Australia and Australian Research Council Centre of Excellence in Structural and Functional Microbial Genomics, Monash University, Clayton Campus,Clayton, VIC, Australia
  • Gabor Juhasz, Department of Anatomy, Cell and Developmental Biology, Eötvös Loránd University, Budapest 1117, Hungary