Sequential Use of Second-Generation Tyrosine Kinase Inhibitor Treatment and Intensive Chemotherapy Induced Long-Term Complete Molecular Response in Imatinib-Resistant CML Patient Presenting as a Myeloid Blast Crisis

<div>Clinical course. The kinetics of the hematological, cytogenetic, and molecular responses to tyrosine kinase inhibitors and conventional chemotherapy seen during the patient’s clinical course are shown. Ara-C: cytosine arabinoside, DNR: daunorubicin, IDR: idarubicin, and ND: not detectable.</div>

Case Reports in Hematology

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Figure 1

Figure 1: Sequential Use of Second-Generation Tyrosine Kinase Inhibitor Treatment and Intensive Chemotherapy Induced Long-Term Complete Molecular Response in Imatinib-Resistant CML Patient Presenting as a Myeloid Blast Crisis 

Figure 1 | Sequential Use of Second-Generation Tyrosine Kinase Inhibitor Treatment and Intensive Chemotherapy Induced Long-Term Complete Molecular Response in Imatinib-Resistant CML Patient Presenting as a Myeloid Blast Crisis 