Amiloride as an Alternate Adjuvant Antiproteinuric Agent in Fabry Disease: The Potential Roles of Plasmin and uPAR

<table>Interactions between globotriaosylceramide (GL-3), uPAR, plasminogen/plasmin, and integrin <i>α</i>v<i>β</i>3 and the development of proteinuria in Fabry disease, counteracted by amiloride.</table>

Case Reports in Nephrology

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Figure 2

Figure 2: Amiloride as an Alternate Adjuvant Antiproteinuric Agent in Fabry Disease: The Potential Roles of Plasmin and uPAR 