Research Article

Biomarkers of Hemodynamic Stress and Aortic Stiffness after STEMI: A Cross-Sectional Analysis

Table 1


Study population ()
Mean/median/number

Age, years59 ± 10
Female, (%)7 (13)
Body mass index, kg/m227 ± 3
Family history for AMI, (%)12 (22)
Smoking status, (%)25 (46)
Hypertension, (%)44 (81)
Hyperlipidemia, (%)36 (67)
Diabetes mellitus, (%)5 (9)
Pain-to-balloon time, min261 (129–759)
Anterior STEMI, (%)17 (32)
Culprit lesion, (%)
 LAD16 (30)
 LCX10 (18)
 RCA28 (52)
Vessel disease, (%)
 124 (44)
 223 (43)
 37 (13)
Creatinine, mg/dL0.98 ± 0.15
eGFR, mL/min/1.73 m283 ± 15
NT-proBNP, ng/L219 (119–412)
MR-proANP, pmol/L88 (68–128)
MR-proADM, nmol/L0.7 ± 0.2
PWV, m/sec7.2 ± 2.0

AMI = acute myocardial infarction; STEMI = ST-segment elevation myocardial infarction; LAD = left anterior descending artery; LCX = left circumflex artery; RCA = right coronary artery; eGFR = estimated glomerular filtration rate; NT-proBNP = N-terminal pro–B-type natriuretic peptide; MR-proANP = mid-regional pro–A-type natriuretic peptide; MR-proADM = mid-regional proadrenomedullin; PWV = pulse wave velocity.