Electroacupuncture at PC6 (Neiguan) Improves Extracellular Signal-Regulated Kinase Signaling Pathways Through the Regulation of Neuroendocrine Cytokines in Myocardial Hypertrophic Rats
Figure 2
The ultrastructure of myocardium in rats for each group ( each group). Ultrastructure features in normal group ((a) ×4000, (b) ×8000, (c) ×10000), model group ((d) ×4000, (e) ×8000, (f) ×8000), PC6 group ((g) ×6000, (h) ×8000, (i) ×8000), and LI4 group ((j) ×5000, (k) ×8000, (l) ×8000). Cardiocyte and micrangium ((a, d, g, j), ×4000~6000), myofibril, I band, and Z-line ((b, e, h, k), ×8000), intercalated disc ((c,f,i, l), ×8000~10000). Some changes of the ultrastructure: mitochondria swelling ((d, e), cell apoptosis (j), endothelial cell interstitial hyperplasia (d), muscle plasma nets expansion (e), out-sync contraction of contraction band ((e, j), and intercalated disc deformation ((f, i, l). With reference to normal group, compared EA group with model group, EA significantly reduced cardiac muscular tissue damage.
