C-Jun N-Terminal Kinase 2 Promotes Liver Injury via the Mitochondrial Permeability Transition after Hemorrhage and Resuscitation
Figure 5
Decreased 4-hydroxynonenal immunostaining after hemorrhage and resuscitation of JNK2-deficient mice. ROS generation was assessed by 4-hydroxynonenal immunocytochemistry livers at 6 h after sham operation of wildtype mice (a) and after H/R to wildtype- (b) and JNK2-deficient (c) mice, as described in Section 2. Bar is 50 μm. per group.
