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Linked References

1. C. A. Fagot and K. Narayan, “Type 2 diabetes in children,” British Medical Journal, vol. 322, pp. 377–387, 2001.
2. C. Levy-Marchal, S. Arslanian, W. Cutfield et al., “Insulin resistance in children: consensus, perspective, and future directions,” Journal of Clinical Endocrinology and Metabolism, vol. 95, no. 12, pp. 5189–5198, 2010. View at Publisher · View at Google Scholar · View at Scopus
3. J. M. Clark, F. L. Brancati, and A. M. Diehl, “The prevalence and etiology of elevated aminotransferase levels in the United States,” American Journal of Gastroenterology, vol. 98, no. 5, pp. 960–967, 2003. View at Publisher · View at Google Scholar · View at Scopus
4. R. S. Strauss, S. E. Barlow, and W. H. Dietz, “Prevalence of abnormal serum aminotransferase values in overweight and obese adolescents,” Journal of Pediatrics, vol. 136, no. 6, pp. 727–733, 2000. View at Scopus
5. A. O. Rantala, M. Lilja, H. Kauma, M. J. Savolainen, A. Reunanen, and Y. A. Kesäniemi, “Gamma-glutamyl transpeptidase and the metabolic syndrome,” Journal of Internal Medicine, vol. 248, no. 3, pp. 230–238, 2000. View at Publisher · View at Google Scholar · View at Scopus
6. I. J. Perry, S. G. Wannamethee, and A. G. Shaper, “Prospective study of serum γ-glutamyltransferase and risk of NIDDM,” Diabetes Care, vol. 21, no. 5, pp. 732–737, 1998. View at Publisher · View at Google Scholar · View at Scopus
7. D. H. Lee, D. R. Jacobs, M. Gross et al., “γ-glutamyltransferase is a predictor of incident diabetes and hypertension: the Coronary Artery Risk Development in Young Adults (CARDIA) study,” Clinical Chemistry, vol. 49, no. 8, pp. 1358–1366, 2003. View at Publisher · View at Google Scholar · View at Scopus
8. B. Vozarova, N. Stefan, R. S. Lindsay et al., “High alanine aminotransferase is associated with decreased hepatic insulin sensitivity and predicts the development of type 2 diabetes,” Diabetes, vol. 51, no. 6, pp. 1889–1895, 2001. View at Scopus
9. A. J. G. Hanley, K. Williams, A. Festa et al., “Elevations in markers of liver injury and risk of type 2 diabetes: the insulin resistance atherosclerosis study,” Diabetes, vol. 53, no. 10, pp. 2623–2632, 2004. View at Publisher · View at Google Scholar · View at Scopus
10. M. Nannipieri, C. Gonzales, S. Baldi et al., “Liver enzymes, the metabolic syndrome, and incident diabetes: the Mexico City diabetes study,” Diabetes Care, vol. 28, no. 7, pp. 1757–1762, 2005. View at Publisher · View at Google Scholar · View at Scopus
11. R. K. Schindhelm, M. Diamant, J. M. Dekker, M. E. Tushuizen, T. Teerlink, and R. J. Heine, “Alanine aminotransferase as a marker of non-alcoholic fatty liver disease in relation to type 2 diabetes mellitus and cardiovascular disease,” Diabetes/Metabolism Research and Reviews, vol. 22, no. 6, pp. 437–443, 2006. View at Publisher · View at Google Scholar · View at Scopus
12. Y. Chang, S. Ryu, E. Sung, and Y. Jang, “Higher concentrations of alanine aminotransferase within the reference interval predict nonalcoholic fatty liver disease,” Clinical Chemistry, vol. 53, no. 4, pp. 686–692, 2007. View at Publisher · View at Google Scholar · View at Scopus
13. R. M. O'Brien and D. K. Granner, “Regulation of gene expression by insulin,” Biochemical Journal, vol. 278, pp. 609–619, 1991.
14. E. Nemesanszky and J. A. Lott, “Gamma-glutamyltransferase and its isoenzymes: progress and problems,” Clinical Chemistry, vol. 31, no. 6, pp. 797–803, 1985. View at Scopus
15. D. R. Karp, K. Shimooku, and P. E. Lipsky, “Expression of γ-glutamyl transpeptidase protects ramos B cells from oxidation-induced cell death,” The Journal of Biological Chemistry, vol. 276, no. 6, pp. 3798–3804, 2001. View at Publisher · View at Google Scholar · View at Scopus
16. M. H. Hanigan and W. A. Ricketts, “Extracellular glutathione is a source of cysteine for cells that express γ-glutamyl transpeptidase,” Biochemistry, vol. 32, no. 24, pp. 6302–6306, 1993. View at Scopus
17. The Korean Pediatric Society Health Statistics Committee, Standard Data Regarding Physical Development of Korean Children and Teenages, The Korean Pediatric Society, Seoul, Republic of Korea, 1999.
18. B. K. Cho, J. H. Kang, J. S. Lee, and B. Y. Yu, “The usefulness of inbody 720 and anthropometric measurement compared with dual-energy x-ray absorptiometry as a diagnostic tool of childhood obesity,” Journal of the Korean Academy of Family Medicine, vol. 28, pp. 523–531, 2007.
19. Y. Copieters and F. Parent, “Blood pressure measurement in epidemiological investigation in teenagers,” European Journal of Epidemiology, vol. 17, no. 10, pp. 901–906, 2001.
20. D. R. Matthews, J. P. Hosker, A. S. Rudenski, B. A. Naylor, D. F. Treacher, and R. C. Turner, “Homeostasis model assessment: insulin resistance and β-cell function from fasting plasma glucose and insulin concentrations in man,” Diabetologia, vol. 28, no. 7, pp. 412–419, 1985. View at Scopus
21. E. Ortega, J. Koska, A. D. Salbe, P. A. Tataranni, and J. C. Bunt, “Serum γ-glutamyl transpeptidase is a determinant of insulin resistance independently of adiposity in Pima Indian children,” Journal of Clinical Endocrinology and Metabolism, vol. 91, no. 4, pp. 1419–1422, 2006. View at Publisher · View at Google Scholar · View at Scopus
22. W. C. Knowler, P. H. Bennett, R. F. Hammam, and M. Miller, “Diabetes incidence and prevalence in Pima Indians: a 19-fold greater incidence than in Rochester, Minnesota,” American Journal of Epidemiology, vol. 108, no. 6, pp. 497–505, 1979. View at Scopus
23. L. J. Baier and R. L. Hanson, “Genetic studies of the etiology of type 2 diabetes in Pima Indians: hunting for pieces to a complicated puzzle,” Diabetes, vol. 53, no. 5, pp. 1181–1186, 2004. View at Publisher · View at Google Scholar · View at Scopus
24. M. I. Goran and B. A. Gower, “Longitudinal study on pubertal insulin resistance,” Diabetes, vol. 50, no. 7–12, pp. 2444–2450, 2001. View at Scopus
25. J. B. Whitfield, “Gamma-glutamyltransferase,” Critical Reviews in Clinical Laboratory Sciences, vol. 38, pp. 263–3553, 2001.
26. O. Nilssen, O. H. Forde, and T. Brenn, “The Tromso study: distribution and population determinants of gamma-glutamyltransferase,” American Journal of Epidemiology, vol. 132, no. 2, pp. 318–326, 1990. View at Scopus
27. A. Paolicchi, M. Emdin, E. Ghliozeni et al., “Human atherosclerotic plaques contain gamma-glutamyl transpeptidase enzyme activity,” Circulation, vol. 109, no. 11, article 1440, 2004. View at Publisher · View at Google Scholar · View at Scopus
28. D. H. Lee and D. R. Jacobs Jr., “Association between serum gamma-glutamyltransferase and C-reactive protein,” Atherosclerosis, vol. 178, no. 2, pp. 327–330, 2005. View at Publisher · View at Google Scholar · View at Scopus
29. J. Yamada, H. Tomiyama, M. Yambe et al., “Elevated serum levels of alanine aminotransferase and gamma glutamyltransferase are markers of inflammation and oxidative stress independent of the metabolic syndrome,” Atherosclerosis, vol. 189, no. 1, pp. 198–205, 2006. View at Publisher · View at Google Scholar · View at Scopus
30. M. D. Michael, R. N. Kulkarni, C. Postic et al., “Loss of insulin signaling in hepatocytes leads to severe insulin resistance and progressive hepatic dysfunction,” Molecular Cell, vol. 6, no. 1, pp. 87–97, 2000. View at Scopus
31. J. B. Schwimmer, R. Deutsch, J. B. Rauch, C. Behling, R. Newbury, and J. E. Lavine, “Obesity, insulin resistance, and other clinicopathological correlates of pediatric nonalcoholic fatty liver disease,” Journal of Pediatrics, vol. 143, no. 4, pp. 500–505, 2003. View at Publisher · View at Google Scholar · View at Scopus
32. G. Marchesini, M. Brizi, G. Blanchi et al., “Nonalcoholic fatty liver disease: a feature of the metabolic syndrome,” Diabetes, vol. 50, no. 8, pp. 1844–1850, 2001. View at Scopus
33. D. A. Patel, S. R. Srinivasan, J. H. Xu, W. Chen, and G. S. Berenson, “Persistent elevation of liver function enzymes within the reference range is associated with increased cardiovascular risk in young adults: the Bogalusa Heart study,” Metabolism, vol. 56, no. 6, pp. 792–798, 2007. View at Publisher · View at Google Scholar · View at Scopus
34. Y. H. Kang, H. K. Min, S. M. Son, I. J. Kim, and Y. K. Kim, “The association of serum gamma glutamyltransferase with components of the metabolic syndrome in the Korean adults,” Diabetes Research and Clinical Practice, vol. 77, no. 2, pp. 306–313, 2007. View at Publisher · View at Google Scholar · View at Scopus
35. A. R. Sinaiko, J. Steinberger, A. Moran et al., “Relation of body mass index and insulin resistance to cardiovascular risk factors, inflammatory factors, and oxidative stress during adolescence,” Circulation, vol. 111, no. 15, pp. 1985–1991, 2005. View at Publisher · View at Google Scholar · View at Scopus
36. J. Steinberger, C. Moorehead, V. Katch, and A. P. Rocchini, “Relationship between insulin resistance and abnormal lipid profile in obese adolescents,” Journal of Pediatrics, vol. 126, no. 5 I, pp. 690–695, 1995. View at Publisher · View at Google Scholar · View at Scopus