Etiopathogenesis of Nonalcoholic Steatohepatitis: Role of Obesity, Insulin Resistance and Mechanisms of Hepatotoxicity

<table>Lipid metabolism in liver. Liver FFA pool is derived from uptake of circulating FFAs and de novo synthesis. The FFAs are then either oxidized or esterified into triglycerides. Triglycerides are then released into circulation as VLDL or stored as vacuoles leading to hepatic steatosis. </table>

International Journal of Hepatology

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Figure 1

Figure 1: Etiopathogenesis of Nonalcoholic Steatohepatitis: Role of Obesity, Insulin Resistance and Mechanisms of Hepatotoxicity 