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Volume 2012 (2012), Article ID 728571, 12 pages
Calpain Dysregulation in Alzheimer’s Disease
Department of Cell and Molecular Biology, Feinberg School of Medicine, Northwestern University, 303 E. Chicago Avenue, Ward 8-140, Chicago, IL 60611, USA
Received 26 August 2012; Accepted 12 September 2012
Academic Editors: A. Caceres and B. Lenarcic
Copyright © 2012 Adriana Ferreira. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Citations to this Article [5 citations]
The following is the list of published articles that have cited the current article.
- MaryKate McBrayer, and Ralph A. Nixon, “Lysosome and calcium dysregulation in Alzheimer's disease: partners in crime,” Biochemical Society Transactions, vol. 41, pp. 1495–1502, 2013.
- Kendall R. Walker, and Giuseppina Tesco, “Molecular mechanisms of cognitive dysfunction following traumatic brain injury,” Frontiers In Aging Neuroscience, vol. 5, 2013.
- Ming Chen, and Huey T. Nguyen, “Our â€œenergy-Ca2+ signaling deficitsâ€ hypothesis and its explanatory potential for key features of Alzheimerâ€™s disease,” Frontiers in Aging Neuroscience, vol. 6, 2014.
- Regina M. Graham, John W. Thompson, and Keith A. Webster, “BNIP3 promotes calcium and calpain-dependent cell death,” Life Sciences, vol. 142, pp. 26–35, 2015.
- Andrew F. Teich, Russell E. Nicholls, Daniela Puzzo, Jole Fiorito, Rosa Purgatorio, Mauro Fa’, and Ottavio Arancio, “Synaptic Therapy in Alzheimer’s Disease: A CREB-centric Approach,” Neurotherapeutics, 2015.