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Volume 2014 (2014), Article ID 570178, 5 pages
The Region Centromeric to HLA-C Is a Key Region for Understanding the Phenotypic Variability of Psoriatic Arthritis
1Rheumatology Department, Hospital Universitario Central de Asturias (HUCA), C/Celestino Villamil S/N, 33006 Oviedo, Spain
2Dermatology Department, HUCA, C/Celestino Villamil S/N, 33006 Oviedo, Spain
3Immunology Department, Histocompatibility Unit, HUCA, C/Celestino Villamil S/N, 33006 Oviedo, Spain
4Department of Functional Biology, IUOPA, University of Oviedo, C/Celestino Villamil S/N, 33006 Oviedo, Spain
Received 24 October 2013; Accepted 10 December 2013; Published 30 January 2014
Academic Editors: M. Schmitt-Egenolf and A. Zalewska
Copyright © 2014 Rubén Queiro et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
- D. D. Gladman, C. Antoni, P. Mease, D. O. Clegg, and O. Nash, “Psoriatic arthritis: epidemiology, clinical features, course, and outcome,” Annals of the Rheumatic Diseases, vol. 64, supplement 2, pp. ii14–ii17, 2005.
- A. Zink, K. Thiele, D. Huscher et al., “Healthcare and burden of disease in psoriatic arthritis. A comparison with rheumatoid arthritis and ankylosing spondylitis,” Journal of Rheumatology, vol. 33, no. 1, pp. 86–90, 2006.
- M. Castelino and A. Barton, “Genetic susceptibility factors for psoriatic arthritis,” Current Opinion in Rheumatology, vol. 22, no. 2, pp. 152–156, 2010.
- D. D. Gladman, V. T. Farewell, F. Pellett, C. Schentag, and P. Rahman, “HLA is a candidate region for psoriatic arthritis: evidence for excessive HLA sharing in sibling pairs,” Human Immunology, vol. 64, no. 9, pp. 887–889, 2003.
- S. González, J. Martínez-Borra, J. C. Torre Alonso et al., “The MICA-A9 triplet repeat polymorphism in the trasmembrane region confers additional susceptibility to develop psoriatic arthritis, and it is independent of the association of Cw*0602 in psoriasis,” Arthritis & Rheumatology, vol. 42, pp. 1010–1016, 1999.
- R. Pollock, V. Chandran, J. Barrett et al., “Differential major histocompatibility complex class I chain-related A allele associations with skin and joint manifestations of psoriatic disease,” Tissue Antigens, vol. 77, no. 6, pp. 554–561, 2011.
- R. Queiro-Silva, J. C. Torre-Alonso, T. Tinturé-Eguren, and I. López-Lagunas, “The effect of HLA-DR antigens on the susceptibility to, and clinical expression of psoriatic arthritis,” Scandinavian Journal of Rheumatology, vol. 33, no. 5, pp. 318–322, 2004.
- R. Queiro, S. González, M. Alperi et al., “HLA-DR17 is associated with enthesitis in psoriatic arthritis,” Joint Bone Spine, vol. 78, no. 4, pp. 428–429, 2011.
- W. Taylor, D. Gladman, P. Helliwell, A. Marchesoni, P. Mease, and H. Mielants, “Classification criteria for psoriatic arthritis: development of new criteria from a large international study,” Arthritis & Rheumatism, vol. 54, no. 8, pp. 2665–2673, 2006.
- J. M. H. Moll and V. Wright, “Psoriatic arthritis,” Seminars in Arthritis and Rheumatism, vol. 3, no. 1, pp. 55–78, 1973.
- S. González, J. Martínez-Borra, A. López-Vázquez, S. García-Fernández, J. C. Torre-Alonso, and C. López-Larrea, “MICA rather than MICB, TNFA, or HLA-DRB1 is associated with susceptibility to psoriatic arthritis,” The Journal of Rheumatology, vol. 29, pp. 973–978, 2002.
- G. Tamiya, M. Ota, Y. Katsuyama et al., “Twenty-six new polymorphic microsatellite markers around the HLA-B, -C and -E loci in the human MHC class I region,” Tissue Antigens, vol. 51, no. 4, part 1, pp. 337–346, 1998.
- J. A. Traherne, “Human MHC architecture and evolution: implications for disease association studies,” International Journal of Immunogenetics, vol. 35, no. 3, pp. 179–192, 2008.
- C. López-Larrea, B. Suárez-Alvarez, A. López-Soto, A. López-Vázquez, and S. Gonzalez, “The NKG2D receptor: sensing stressed cells,” Trends in Molecular Medicine, vol. 14, pp. 179–189, 2008.
- S. González, V. Groh, and T. Spies, “Immunobiology of human NKG2D and its ligands,” Current Topics in Microbiology and Immunology, vol. 298, pp. 121–138, 2006.
- R. Queiro, C. Sarasqueta, J. Belzunegui, C. Gonzalez, M. Figueroa, and J. C. Torre-Alonso, “Psoriatic spondyloarthropathy: a comparative study between HLA-B27 positive and HLA-B27 negative disease,” Seminars in Arthritis and Rheumatism, vol. 31, no. 6, pp. 413–418, 2002.
- P. Y. P. C. Ho, A. Barton, J. Worthington, W. Thomson, A. J. Silman, and I. N. Bruce, “HLA-Cw6 and HLA-DRB1*07 together are associated with less severe joint disease in psoriatic arthritis,” Annals of the Rheumatic Diseases, vol. 66, no. 6, pp. 807–811, 2007.
- A. Mameli, A. Cauli, E. Taccari et al., “Association of MICA alleles with psoriatic arthritis and its clinical forms. A multicenter Italian study,” Clinical and Experimental Rheumatology, vol. 26, no. 4, pp. 649–652, 2008.
- D. McGonagle, R. J. U. Lories, A. L. Tan, and M. Benjamin, “The concept of a “synovio-entheseal complex” and its implications for understanding joint inflammation and damage in psoriatic arthritis and beyond,” Arthritis & Rheumatism, vol. 56, no. 8, pp. 2482–2491, 2007.