Diabetes has been considered as a chronic inflammatory disease for decades. Increasing evidence indicates that inflammation plays a critical role in the pathogenesis of both type 1 and type 2 diabetes. Type 1 diabetes is characterized by T cell-mediated autoimmune destruction of pancreatic β cells, while inflammatory responses have been detected in both islets and insulin-sensitive tissues such as adipose tissue, liver, and muscle in type 2 diabetes. This special issue aims at furthering our understanding on the inflammatory regulation in diabetes and metabolic dysfunction.

We invite authors to contribute original research articles as well as review articles that focus on the topic of inflammatory regulation in metabolic dysfunction.

Potential topics include but are not limited to the following:

• Mechanism regulating inflammation in diabetes
• Inflammatory markers in the diagnosis of type 1 or type 2 diabetes
• Clinical immunology in diabetes
• The role of acute phase proteins and cytokines in metabolic regulation
• Relationships between inflammation and metabolism
• Inflammation in organs/tissues (fat, liver, muscle, pancreas, etc.) and insulin resistance
• Anti-inflammatory agents in diabetes
• Insulin and glucose metabolism in the modulation of immune cell function
• The role of metabolites in modulating inflammatory processes
• Antidiabetic drugs and inflammation
• Role of immune cells in the development and progression of diabetes