Sensitization of Cervical Cancer Cells to Cisplatin by Genistein: The Role of NF𝜿B and Akt/mTOR Signaling Pathways

Figure 2

The intensity of the bands was quantified by the densitometric analysis. The expression of (a) NF-κB, (b) p-mTOR, (c) p-p70S6K1, (d) p-4E-BP1, and (e) p-Akt in HeLa cells. Cells untreated or treated with 25 μM genistein, 250 nM cisplatin (Cis), and the combination (genistein + cisplatin). β-actin antibodies were used as internal controls for equal loading of proteins. Data are percent of the control. *𝑃<0.05; **𝑃<0.01.

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