The anomalous posterior vitreous detachment plays a major role in the formation of anterior and tangential epiretinal traction. The presence of vitreoschisis associated with a strong vitreopapillary adhesion of posterior cortex enhances the force of the tangential vector. The outer layer of posterior vitreous cortex leads to tangential traction responsible for the formation of macular hole or macular pucker. Advances in the tomographic analysis of vectors of vitreous traction have highlighted the selected cases that can benefit from a new enzymatic intravitreal treatment providing a different perspective for therapeutic strategies. The surgical treatment, consisting of the vitrectomy and peeling of epiretinal traction and inner limiting membrane, still plays the major role in the treatment of vitreomacular interface. We particularly take an interest in manuscripts that report relevance of vitreous traction associated with anomalous posterior vitreous detachment for improved diagnosis, prognosis, and surgical or enzymatic release of traction.

Potential topics include, but are not limited to:

• Vectors of traction associated with anomalous posterior vitreous detachment
• Tangential traction and enface tomography
• Vitreoschisis of posterior cortex
• Vitreopapillary adhesion
• Idiopathic epiretinal membrane: stages of traction
• Vitreomacular interfaces in diabetic retinopathy
• Surgical treatment and vertical retinal gliosis
• Role of intravitreal ocriplasmin in treatment of vitreomacular traction
• Retinal damage induced by inner limiting membrane and macular peeling