Thimerosal-Derived Ethylmercury Is a Mitochondrial Toxin in Human Astrocytes: Possible Role of Fenton Chemistry in the Oxidation and Breakage of mtDNA

<table>Changes in mitochondrial membrane potential and of ROS generation induced by ethylmercury in Normal Human Astrocytes. Changes in mitotracker and ROS induced DCF levels caused by incubation with increasing concentrations of Thimerosal (b) and the time course of incubation with 14.4 <svg height="9.6750002" id="M27" style="vertical-align:-2.29482pt" version="1.1" viewbox="0 0 9.6374998 9.6750002" width="9.6374998" xmlns="http://www.w3.org/2000/svg">
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</svg>M Thimerosal (a), with respect to control cultures. Cells were imaged in the centerfield of three independent wells, consisting of an average of 44 cells per field, with a standard deviation of 16.5.</table>

Journal of Toxicology

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Figure 1

Figure 1: Thimerosal-Derived Ethylmercury Is a Mitochondrial Toxin in Human Astrocytes: Possible Role of Fenton Chemistry in the Oxidation and Breakage of mtDNA 

Figure 1 | Thimerosal-Derived Ethylmercury Is a Mitochondrial Toxin in Human Astrocytes: Possible Role of Fenton Chemistry in the Oxidation and Breakage of mtDNA 