Long Course Hyperbaric Oxygen Stimulates Neurogenesis and Attenuates Inflammation after Ischemic Stroke

<table class="table-group" width="800px"><tr class="fig-image" id="a"><td><img alt="512978.fig.005a" src="https://static.hindawi.com/articles/mi/volume-2013/512978/figures/512978.fig.005a.jpg"/></td></tr><tr class="fig-caption"><td align="center"><table><tr><td><b>(a) </b>BrdU/GFAP in hippocampus</td></tr></table></td></tr><tr class="fig-image" id="b"><td><img alt="512978.fig.005b" src="https://static.hindawi.com/articles/mi/volume-2013/512978/figures/512978.fig.005b.jpg"/></td></tr><tr class="fig-caption"><td align="center"><table><tr><td><b>(b) </b>BrdU/GFAP in cortex</td></tr></table></td></tr></table>

<table>Long course HBO increased gliosis. ((a) and (b)) Demonstration of double staining showed that there were significantly more newly forming or reactive glia in the ischemic boundary area (dentate gyrus (a) and cortex (b)) of HBO3wks rats than HBO2ds rats. The intensity of co-staining with BrdU and Neu-N was more prominent in HBO3wks group.</table>

Mediators of Inflammation

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Figure 5

Figure 5: Long Course Hyperbaric Oxygen Stimulates Neurogenesis and Attenuates Inflammation after Ischemic Stroke 