Carbon Monoxide Inhibits Tenascin-C Mediated Inflammation via IL-10 Expression in a Septic Mouse Model

<div>Schemata of the proposed CO/IL-10-dependent anti-inflammatory signaling pathway. Carbon monoxide increased IL-10 expression and inhibited TN-C-induced inflammation in a septic mouse model<i> in vivo</i> and<i> in vitro</i>.</div>

Mediators of Inflammation

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Figure 6

Figure 6: Carbon Monoxide Inhibits Tenascin-C Mediated Inflammation via IL-10 Expression in a Septic Mouse Model 