Sensory Neuropeptides and Endogenous Opioids Expression in Human Dental Pulp with Asymptomatic Inflammation: <i>In Vivo</i> Study

<div>Proposed relationship between neurogenic inflammatory and endogenous opioid analgesic mechanisms. SP: substance P, CGRP: calcitonin gene-related peptide, <i>β</i>-End: <i>β</i>-endorphin, Met-Enk: methionine-enkephalin, NK1-R: neurokinin 1 receptor, CGRP1/2-R: calcitonin gene-related peptide receptors 1 and 2, MOR: <i>μ</i>-opioid receptor, DOR: <i>δ</i>-opioid receptor, OEA: opioid endogenous analgesia, AI: asymptomatic inflammation, and PAN: primary afferent neuron.</div>

Mediators of Inflammation

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Figure 1: Sensory Neuropeptides and Endogenous Opioids Expression in Human Dental Pulp with Asymptomatic Inflammation: <i>In Vivo</i> Study 

Mediators of Inflammation

Sensory Neuropeptides and Endogenous Opioids Expression in Human Dental Pulp with Asymptomatic Inflammation: In Vivo Study

Figure 1