N-n-Butyl Haloperidol Iodide Ameliorates Cardiomyocytes Hypoxia/Reoxygenation Injury by Extracellular Calcium-Dependent and -Independent Mechanisms

Table 3

Effects of F₂ on cAMP levels of cardiomyocyte in extracellular-calcium-free H/R (𝑛=7).

Group	cAMP (pmol/mL)
0CaCon	2.82 ± 0.55
0CaH/R	1.60 ± 0.35
0CaH/R + F2	1.48 ± 0.56

F₂, N-n-butyl haloperidol iodide; cAMP: cyclic adenosine monophosphate; H/R: hypoxia/reoxygenation.