[Retracted] Involvement of Nrf2-Mediated Upregulation of Heme Oxygenase-1 in Mollugin-Induced Growth Inhibition and Apoptosis in Human Oral Cancer Cells

<table>Effects of mollugin on cell viability in primary (HN4, (a)), metastatic oral cancer cells (HN12, (b)), skin keratinocytes (HaCaT, (c)), and gingival fibroblasts (d) as measured by MTT assay. *Statistically significant difference as compared to control, <svg height="11.375" id="M3" style="vertical-align:-0.1638pt" version="1.1" viewbox="0 0 58.712502 11.375" width="58.712502" xmlns="http://www.w3.org/2000/svg" xmlns:xlink="http://www.w3.org/1999/xlink">
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</svg>. The same experiment was repeated in three separate cultures, and the data were presented as the means ± SD of five observations.</table>

BioMed Research International

fig1

Figure 1

Figure 1: [Retracted] Involvement of Nrf2-Mediated Upregulation of Heme Oxygenase-1 in Mollugin-Induced Growth Inhibition and Apoptosis in Human Oral Cancer Cells 

Figure 1 | [Retracted] Involvement of Nrf2-Mediated Upregulation of Heme Oxygenase-1 in Mollugin-Induced Growth Inhibition and Apoptosis in Human Oral Cancer Cells 