Therapeutic Implications of Estrogen for Cerebral Vasospasm and Delayed Cerebral Ischemia Induced by Aneurysmal Subarachnoid Hemorrhage

<table>Pathways of E2-mediated facilitation of vasodilation and inhibition of vasoconstriction. E2: 17<i>β</i>-estradiol, eNOS: endothelial nitric oxide synthase, ET-1: endothelin-1, iNOS: inducible nitric oxide synthase, NO: nitric oxide, and ROS: reactive oxygen species.</table>

BioMed Research International

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Figure 1

Figure 1: Therapeutic Implications of Estrogen for Cerebral Vasospasm and Delayed Cerebral Ischemia Induced by Aneurysmal Subarachnoid Hemorrhage 