Dihydromyricetin Attenuates TNF-α-Induced Endothelial Dysfunction through miR-21-Mediated DDAH1/ADMA/NO Signal Pathway

Figure 1

DMY represses TNF-α-induced miR-21 expression in HUVECs. (a) The effect of different doses of DMY on HUVECs viability; (b) TNF-α induces miR-21 expression in a time-dependent manner; (c) DMY represses TNF-α-induced miR-21 expression in a dose-dependent manner. Data was expressed as mean ± SD, = 3–6, versus control, versus control, versus TNF-α, and versus TNF-α.

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