Research Article

tRNA-Derived Fragments in Podocytes with Adriamycin-Induced Injury Reveal the Potential Mechanism of Idiopathic Nephrotic Syndrome

Figure 1

Experimental procedure and establishment of the podocyte injury model. (a) Experimental procedure. (b) Podocyte differentiation. Compared with those in undifferentiated podocytes, nephrin and podocin protein expression levels in differentiated podocytes were significantly upregulated. (c) Podocyte injury. The protein level of cleaved caspase 3 was significantly upregulated in the Adr group.
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