Research Article

Silencing Cardiac Troponin I-Interacting Kinase Reduces Lipopolysaccharide-Induced Sepsis-Induced Myocardial Dysfunction in Rat by Regulating Apoptosis-Related Proteins

Figure 1

TNNI3K was high expressed in LPS-induced H9C2 cells. (a) The mRNA level of TNNI3K in control group and LPS group. (b) The protein level of TNNI3K in the control group and LPS group. indicates comparison with the control group. LPS: lipopolysaccharide; TNNI3K: troponin I-interacting kinase.
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