Ischemic Postconditioning (IPostC) Protects Fibrotic and Cirrhotic Rat Livers after Warm Ischemia

Table 1

Histological evaluation of healthy, fibrotic, and cirrhotic livers after warm ischemia ± IPostC.

Liver model	IRI-parameter	Comments
Healthy	Fibrosis: 0
	Inflammation: 1	single cell necroses; apoptosis; mitoses
	Bile duct proliferation: 0
	Fat deposition: 2	microvesicular
	Group necroses: 2	diffuse; cells without clear core loss, but with loss of color; loosened cytoplasm
BDL	Fibrosis: 1	septa in the area of bile duct proliferation
	Inflammation: 2	Single cell necroses; apoptosis; mitoses
	Bile duct proliferation: 3	diffusely distributed throughout the parenchyma
	Fat deposition: 1	very low microvesicular
	Group necroses: 1
TAA	Fibrosis: 2	proliferation of fibroblasts; focally confluent septa
	Inflammation: 2	mixed; predominantly chronic focal suppurative-destructive cholangitis; single cell necroses; apoptosis; mitoses
	Bile duct proliferation: 2	plus accumulation of proliferations of vessels and fibroblasts
	Fat deposition: 1	very low microvesicular
	Group necroses: 1	focally with beginning connective tissue remodeling