Table 1: Histological evaluation of healthy, fibrotic, and cirrhotic livers after warm ischemia ± IPostC.

Liver modelIRI-parameterComments

HealthyFibrosis: 0
Inflammation: 1single cell necroses; apoptosis; mitoses
Bile duct proliferation: 0
Fat deposition: 2microvesicular
Group necroses: 2diffuse; cells without clear core loss, but with loss of color; loosened cytoplasm

BDLFibrosis: 1septa in the area of bile duct proliferation
Inflammation: 2Single cell necroses; apoptosis; mitoses
Bile duct proliferation: 3diffusely distributed throughout the parenchyma
Fat deposition: 1very low microvesicular
Group necroses: 1

TAAFibrosis: 2proliferation of fibroblasts; focally confluent septa
Inflammation: 2mixed; predominantly chronic focal suppurative-destructive cholangitis; single cell necroses; apoptosis; mitoses
Bile duct proliferation: 2plus accumulation of proliferations of vessels and fibroblasts
Fat deposition: 1very low microvesicular
Group necroses: 1focally with beginning connective tissue remodeling