Case Report

A New Case of Spontaneous Regression of Inflammatory Hepatic Pseudotumor

Table 1

Imaging features of common hepatic lesions.

USCT scanMRI

Hemangioma(i) hyperechoic(i) low attenuation on the noncontrast CThyperintense on T2
(ii) well-defined or lobulated borders(ii) peripheral nodular enhancement

Focal nodular hyperplasia (FNH)(i) isoechoicearly enhancement with characteristic central scarhypointense with central scare which is hyperintense on T2
(ii) well-defined borders

Hepatic adenomadiscretely hypoechoicearly enhancementhyperintense on T1 and T2

Hepatocellular carcinoma(i) hypoechoic, heterogeneous(i) early and heterogeneous enhancement hypointense on T1 and hyperintense on T2
(ii) portal thrombosis(ii) hypodense, with only the capsule enhancing, on delayed-phase
(iii) portal vein occlusion from venous invasion with intraluminal tumor present and expansion of the vessel

Metastatic disease(i) multiple hypoechoic lesions thick and irregular rim with enhancement(i) mildly hyperintense to liver
(ii) lesions with a hypoechoic rim or halo pattern(ii) irregular or rim enhancement

IPT of the liver(i) usually unique(i) low attenuation on the noncontrast CT(i) hypointense on T1, hyperintense on T2
(ii) hypoechoic(ii) late and peripheral enhancement(ii) heterogenic enhancement
(iii) well-defined borders(iii) venous occlusion from gross thickening of the wall of the vein