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Case Reports in Ophthalmological Medicine
Volume 2016, Article ID 3806056, 5 pages
http://dx.doi.org/10.1155/2016/3806056
Case Report

Pediatric Balint’s Syndrome Variant: A Possible Diagnosis in Children

1Department of Ophthalmology, The Cerebral Visual Impairment Clinic, Christian Medical College and Hospital, Vellore, Tamil Nadu 632001, India
2Department of Radiodiagnosis, Christian Medical College, Vellore, Tamil Nadu, India
3Department of Vision Sciences, Glasgow Caledonian University, Cowcaddens Road, Glasgow G4 0BA, UK

Received 13 July 2016; Accepted 19 October 2016

Academic Editor: Stephen G. Schwartz

Copyright © 2016 Swetha Sara Philip et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Abstract

Balint’s syndrome is well described in adults, but not in children. It is caused by bilateral posterior parietal lobe damage and comprises a triad of simultanagnosia (inability to simultaneously see more than a small number of items), optic ataxia (impaired visual guidance of movement of the limbs and body), and apraxia of gaze (inability to volitionally direct gaze despite the requisite motor substrate) often associated with homonymous lower visual field loss. We, here, describe five children (four males, one female; mean age 7.4 years, [range 4−11 years]; birth weight ≤ 2.5 kg; four were born ≤ 36 weeks of gestational age and one at 40 weeks) who presented to the Cerebral Visual Impairment Clinic at a tertiary care center in South India with clinical features remarkably consistent with the above description. In all children neuroimaging showed bilateral parietooccipital gliosis with regional white matter volume loss and focal callosal thinning, consistent with perinatal hypoxic ischemic encephalopathy and possible neonatal hypoglycemia.