Research Article
CXCL16 Induces the Progression of Pulmonary Fibrosis through Promoting the Phosphorylation of STAT3
Figure 6
The antibody of CXCL16 reduced the effect of LPS on mouse LF cells. (a) The antibody of CXCL16 deeply suppressed the production of HYP in LPS cultured cells ( vs Blank; vs L100). (b) The phosphorylation of STAT3 was inhibited by the antibody of CXCL16 in LPS cultured cells ( vs Blank, vs Blank; vs L100, vs L100).
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