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Disease Markers
Volume 2015, Article ID 828145, 11 pages
http://dx.doi.org/10.1155/2015/828145
Research Article

Identification of Differentially Expressed Genes Associated with Prognosis of B Acute Lymphoblastic Leukemia

1Departamento de Bioquimica y Medicina Molecular, Facultad de Medicina, Universidad Autonoma de Nuevo Leon, Avenida F. I. Madero, S/N, Col. Mitras Centro, 64460 Monterrey, NL, Mexico
2Molecular Medicine Laboratory, Unidad Academica de Medicina Humana y Ciencias de la Salud, Universidad Autonoma de Zacatecas, Carretera Zacatecas-Guadalajara Km 6, 98160 Ejido la Escondida, ZAC, Mexico
3Servicio de Hematologia, Hospital Universitario “Jose Eleuterio Gonzalez”, Universidad Autonoma de Nuevo Leon, Avenida F. I. Madero, S/N, Col. Mitras Centro, 64460 Monterrey, NL, Mexico
4Instituto Nacional de Medicina Genomica (INMEGEN), Periferico Sur No. 4809, Col. Arenal Tepepan, Delegacion Tlalpan, 14610 Mexico, DF, Mexico
5Mesoamerican Center of Public Health Studies and Disasters (CEMESAD), Universidad Autonoma de Chiapas (UNACH), Avenida Pista Principal esq Pista Secundaria, S/N, 30798 Tapachula, CHIS, Mexico
6Centro de Investigacion y Desarrollo en Ciencias de la Salud, Universidad Autonoma de Nuevo Leon, Carlos Canseco, S/N, Col. Mitras Centro, 64460 Monterrey, NL, Mexico

Received 31 October 2014; Revised 27 January 2015; Accepted 30 January 2015

Academic Editor: Olav Lapaire

Copyright © 2015 Idalia Garza-Veloz et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Supplementary Material

Supplementary Material includes the details of the treatment of B-ALL patients involved in the study. This is shown in Additional Table 1. The classification of 45 genes evaluated by cellular signalling pathway and general characteristics of the 47 sets of primers used can be seen in Additional Table 2, while the comparisons of ΔCq values between B-ALL cases and healthy controls in each of the different times studied comes in Additional Table 3. Finally, the descriptive statistics of expression levels for the 30 genes with differences in at least one time between cases and controls may be found in Additional Table 4. All the references for the Supplementary Material, including those related with the previous studies of individual correlations between the 45 genes and cancer were added at the end of the document.

  1. Supplementary Material