Regulatory Mechanism of LINC00152 on Aggravating Heart Failure through Triggering Fibrosis in an Infarcted Myocardium

<div>LINC00152 promoted proliferative and migratory abilities in cardiac fibroblasts. (a) Transfection of either si-LINC00152-1# or si-LINC00152-2# significantly downregulated LINC00152 in primary cardiac fibroblasts. (b) Knockdown of LINC00152 weakened proliferation in primary cardiac fibroblasts. (c) Knockdown of LINC00152 weakened migration in primary cardiac fibroblasts (magnification: 200x).</div>

Disease Markers

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Figure 3

Figure 3: Regulatory Mechanism of LINC00152 on Aggravating Heart Failure through Triggering Fibrosis in an Infarcted Myocardium 