Salidroside Improves Homocysteine-Induced Endothelial Dysfunction by Reducing Oxidative Stress
The effect of the expression of NOX2 and NOX4 in rat aortas. (a) Homocysteine (Hcy) increased the NOX2 expression which was partially reversed by salidroside and DPI but not tiron. (b) The NOX4 expression was unchanged in all treatment groups. Results are means ± SEM of 4 separate experiments. * versus control; # versus Hcy.
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