Salidroside Improves Homocysteine-Induced Endothelial Dysfunction by Reducing Oxidative Stress
ROS accumulation in aortas (a) and in primary endothelial cells (b) as detected by DHE staining. Homocysteine- (Hcy-) stimulated increase was inhibited by salidroside, tiron, and DPI in ROS in both preparations. Results are means ± SEM of 5 separate experiments. * versus control; # versus Hcy.
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