Table of Contents Author Guidelines Submit a Manuscript
Evidence-Based Complementary and Alternative Medicine
Volume 2013, Article ID 828754, 7 pages
http://dx.doi.org/10.1155/2013/828754
Research Article

A Brief Analysis of Traditional Chinese Medical Elongated Needle Therapy on Acute Spinal Cord Injury and Its Mechanism

1Research Institute of Acupuncture and Moxibustion, Xiaoshan Traditional Chinese Medical Hospital, Zhejiang 311200, China
2Research Institute of Acupuncture and Moxibustion, Zhejiang Chinese Medical University, Hangzhou 31012, China
3Research Institute of Acupuncture and Moxibustion, Medical College of Xiamen University, Xiamen 361005, China
4Department of Acupuncture and Moxibustion, The Second Affiliated Hospital, Medical College of Zhejiang University, Hangzhou 310009, China

Received 26 September 2013; Revised 22 October 2013; Accepted 22 October 2013

Academic Editor: Calvin Yu-Chian Chen

Copyright © 2013 Mengxuan Du et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Linked References

  1. G. T. Williams and C. A. Smith, “Molecular regulation of apoptosis: genetic controls on cell death,” Cell, vol. 74, no. 5, pp. 777–779, 1993. View at Publisher · View at Google Scholar · View at Scopus
  2. I. Heo, B.-C. Shin, Y.-D. Kim, E.-H. Hwang, C. W. Han, and K.-H. Heo, “Acupuncture for spinal cord injury and its complications: a systematic review and meta-analysis of randomized controlled trials,” Evidence-Based Complementary and Alternative Medicine, vol. 2013, Article ID 364216, 18 pages, 2013. View at Publisher · View at Google Scholar
  3. S.-F. Huang, Y. Ding, J.-W. Ruan et al., “An experimental electro-acupuncture study in treatment of the rat demyelinated spinal cord injury induced by ethidium bromide,” Neuroscience Research, vol. 70, no. 3, pp. 294–304, 2011. View at Publisher · View at Google Scholar · View at Scopus
  4. L. Mao, “Analysis on spectrum of using elongated needle therapy to curing diseases,” Chinese Medicine Research, vol. 22, no. 1, pp. 61–62, 2009. View at Google Scholar
  5. E. D. Crown, Z. Ye, K. M. Johnson, G.-Y. Xu, D. J. McAdoo, and C. E. Hulsebosch, “Increases in the activated forms of ERK 1/2, p38 MAPK, and CREB are correlated with the expression of at-level mechanical allodynia following spinal cord injury,” Experimental Neurology, vol. 199, no. 2, pp. 397–407, 2006. View at Publisher · View at Google Scholar · View at Scopus
  6. C. E. Hulsebosch, B. C. Hains, E. D. Crown, and S. M. Carlton, “Mechanisms of chronic central neuropathic pain after spinal cord injury,” Brain Research Reviews, vol. 60, no. 1, pp. 202–213, 2009. View at Publisher · View at Google Scholar · View at Scopus
  7. H. Li, H. Zhu, C.-J. Xu, and J. Yuan, “Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis,” Cell, vol. 94, no. 4, pp. 491–501, 1998. View at Google Scholar · View at Scopus
  8. K. R. Byrnes, B. A. Stoica, S. Fricke, S. di Giovanni, and A. I. Faden, “Cell cycle activation contributes to post-mitotic cell death and secondary damage after spinal cord injury,” Brain, vol. 130, no. 11, pp. 2977–2992, 2007. View at Publisher · View at Google Scholar · View at Scopus
  9. K. Nakamura, E. Bossy-Wetzel, K. Burns et al., “Changes in endoplasmic reticulum luminal environment affect cell sensitivity to apoptosis,” Journal of Cell Biology, vol. 150, no. 4, pp. 731–740, 2000. View at Google Scholar
  10. D. Siniscalco, C. Fuccio, C. Giordano et al., “Role of reactive oxygen species and spinal cord apoptotic genes in the development of neuropathic pain,” Pharmacological Research, vol. 55, no. 2, pp. 158–166, 2007. View at Publisher · View at Google Scholar · View at Scopus
  11. R. V. Rao, E. Hermel, S. Castro-Obregon et al., “Coupling endoplasmic reticulum stress to the cell death program. Mechanism of caspase activation,” Journal of Biological Chemistry, vol. 276, no. 36, pp. 33869–33874, 2001. View at Publisher · View at Google Scholar · View at Scopus
  12. W. R. Yu and M. G. Fehlings, “Fas/FasL-mediated apoptosis and inflammation are key features of acute human spinal cord injury: implications for translational, clinical application,” Acta Neuropathologica, vol. 122, no. 6, pp. 747–761, 2011. View at Publisher · View at Google Scholar · View at Scopus
  13. K. Kandasamy, S. M. Srinivasula, E. S. Alnemri et al., “Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release,” Cancer Research, vol. 63, no. 7, pp. 1712–1721, 2003. View at Google Scholar · View at Scopus
  14. R. Eguchi, S. Toné, A. Suzuki et al., “Possible involvement of caspase-6 and -7 but not caspase-3 in the regulation of hypoxia-induced apoptosis in tube-forming endothelial cells,” Experimental Cell Research, vol. 315, no. 2, pp. 327–335, 2009. View at Publisher · View at Google Scholar · View at Scopus
  15. H. Khalil, N. Peltzer, J. Walicki et al., “Caspase-3 protects stressed organs against cell death,” Molecular and Cellular Biology, vol. 32, no. 22, pp. 4523–4533, 2012. View at Publisher · View at Google Scholar
  16. B. C. Shin, M. S. Lee, J. C. Kong, and I. Jang, “Acupuncture for spinal cord injury survivors in Chinese literature: a systematic review,” Complementary Therapies in Medicine, vol. 17, no. 5, pp. 316–327, 2009. View at Publisher · View at Google Scholar