Protective Effects of Salidroside on Mitochondrial Functions against Exertional Heat Stroke-Induced Organ Damage in the Rat

Figure 5

Salidroside inhibited excessive generation of ROS triggered by EHS. (a) ROS detected using specific fluorescent probes in hepatocytes and cardiomyocytes in the control group, high temperature group, exercise group, EHS group, and EHS + salidroside group (20, 50, and 100 μM). (b) ROS level in hepatocytes in the control group, high temperature group, exercise group, EHS group, and EHS + salidroside group (20, 50, and 100 μM). (c) ROS level in cardiomyocytes in the control group, high temperature group, exercise group, EHS group, and EHS + salidroside group (20, 50, and 100 μM). Results were expressed as the mean ± SEM. . , and .

(a)

(b)

(c)