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Gastroenterology Research and Practice
Volume 2012 (2012), Article ID 473960, 5 pages
Is Post-ERCP Pancreatitis a Genetically Predisposed Complication?
1Gastrointestinal Endoscopy Unit, Democritus University of Thrace, University General Hospital of Alexandroupolis, 68100 Dragana, Greece
2Department of Medical Laboratories, Alexandreio Technological Institute of Thessaloniki, 57400 Thessaloniki, Greece
3Department of Radiology, Democritus University of Thrace, University General Hospital of Alexandroupolis, 68100 Dragana, Greece
41st Surgical Department, Democritus University of Thrace, University General Hospital of Alexandroupolis, 68100 Dragana, Greece
5Gastroenterology Department, George Papanikolaou Hospital, 57010 Thessaloniki, Greece
Received 16 February 2012; Revised 19 June 2012; Accepted 9 July 2012
Academic Editor: Grazyna Rydzewska
Copyright © 2012 Konstantinos Mystakidis et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
- S. Sherman and G. A. Lehman, “ERCP- and endoscopic sphincterotomy-induced pancreatitis,” Pancreas, vol. 6, no. 3, pp. 350–367, 1991.
- T. Mazaki, H. Masuda, and T. Takayama, “Prophylactic pancreatic stent placement and post-ERCP pancreatitis: a systematic review and meta-analysis,” Endoscopy, vol. 42, no. 10, pp. 842–852, 2010.
- E. Masci, A. Mariani, S. Curioni, and P. A. Testoni, “Risk factors for pancreatitis following endoscopic retrograde cholangiopancreatography: a meta-analysis,” Endoscopy, vol. 35, no. 10, pp. 830–834, 2003.
- M. L. Freeman and N. M. Guda, “Prevention of post-ERCP pancreatitis: a comprehensive review,” Gastrointestinal Endoscopy, vol. 59, no. 7, pp. 845–864, 2004.
- S. T. Cooper and A. Slivka, “Incidence, risk factors, and prevention of post-ERCP pancreatitis,” Gastroenterology Clinics of North America, vol. 36, no. 2, pp. 259–276, 2007.
- C. L. Cheng, S. Sherman, J. L. Watkins et al., “Risk factors for post-ERCP pancreatitis: a prospective multicenter study,” American Journal of Gastroenterology, vol. 101, no. 1, pp. 139–147, 2006.
- M. L. Freeman and N. M. Guda, “30 years of ERCP and still the same problems?” Endoscopy, vol. 39, no. 9, pp. 833–835, 2007.
- P. Wang, Z. S. Li, F. Liu et al., “Risk factors for ERCP-related complications: a prospective multicenter study,” American Journal of Gastroenterology, vol. 104, no. 1, pp. 31–40, 2009.
- P. B. Cotton, D. A. Garrow, J. Gallagher, and J. Romagnuolo, “Risk factors for complications after ERCP: a multivariate analysis of 11,497 procedures over 12 years,” Gastrointestinal Endoscopy, vol. 70, no. 1, pp. 80–88, 2009.
- P. A. Testoni, A. Mariani, A. Giussani et al., “Risk factors for post-ERCP pancreatitis in high-and low-volume centers and among expert and non-expert operators: a prospective multicenter study,” American Journal of Gastroenterology, vol. 105, no. 8, pp. 1753–1761, 2010.
- D. C. Whitcomb, M. C. Gorry, R. A. Preston et al., “Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene,” Nature Genetics, vol. 14, no. 2, pp. 141–145, 1996.
- J. M. Chen, B. Mercier, M. P. Audrezet, and C. Ferec, “Mutational analysis of the human pancreatic secretory trypsin inhibitor (PSTI) gene in hereditary and sporadic chronic pancreatitis,” Journal of Medical Genetics, vol. 37, no. 1, pp. 67–69, 2000.
- H. Witt, W. Luck, H. C. Hennies et al., “Mutations in the gene encoding the serine protease inhibitor, Kazal type 1 are associated with chronic pancreatitis,” Nature Genetics, vol. 25, no. 2, pp. 213–216, 2000.
- R. H. Pfützer, M. M. Barmada, A. P. Brunskill et al., “SPINK1/PSTI polymorphisms act as disease modifiers in familial and idiopathic chronic pancreatitis,” Gastroenterology, vol. 119, no. 3, pp. 615–623, 2000.
- P. B. Cotton, G. Lehman, J. Vennes et al., “Endoscopic sphincterotomy complications and their management: an attempt at consensus,” Gastrointestinal Endoscopy, vol. 37, no. 3, pp. 383–393, 1991.
- J. P. H. Drenth, R. te Morsche, and J. B. Jansen, “Mutations in serine protease inhibitor Kazal type 1 are strongly associated with chronic pancreatitis,” Gut, vol. 50, no. 5, pp. 687–692, 2002.
- J. Threadgold, W. Greenhalf, I. Ellis et al., “The N34S mutation of SPINK1 (PSTI) is associated with a familial pattern of idiopathic chronic pancreatitis but does not cause the disease,” Gut, vol. 50, no. 5, pp. 675–681, 2002.
- L. Rossi, R. H. Pfützer, S. Parvin et al., “SPINK1/PSTI mutations are associated with tropical pancreatitis in Bangladesh: a preliminary report,” Pancreatology, vol. 1, no. 3, pp. 242–245, 2001.
- G. R. Chandak, M. M. Idris, D. N. Reddy, S. Bhaskar, P. V. Sriram, and L. Singh, “Mutations in the pancreatic secretory trypsin inhibitor gene (PSTI/SPINK1) rather than the cationic trypsinogen gene (PRSS1) are significantly associated with tropical calcific pancreatitis,” Journal of Medical Genetics, vol. 39, no. 5, pp. 347–351, 2002.
- Z. Hassan, V. Mohan, L. Ali et al., “SPINK1 is a susceptibility gene for fibrocalculous pancreatic diabetes in subjects from the Indian subcontinent,” American Journal of Human Genetics, vol. 71, no. 4, pp. 964–968, 2002.
- E. Bhatia, G. Choudhuri, S. S. Sikora et al., “Tropical calcific pancreatitis: strong association with SPINK1 trypsin inhibitor mutations,” Gastroenterology, vol. 123, no. 4, pp. 1020–1025, 2002.
- A. Schneider, A. Suman, L. Rossi et al., “SPINK1/PSTI mutations are associated with tropical pancreatitis and type II diabetes mellitus in Bangladesh,” Gastroenterology, vol. 123, no. 4, pp. 1026–1030, 2002.
- H. Witt, W. Luck, M. Becket et al., “Mutation in the SPINK1 trypsin inhibitor gene, alcohol use, and chronic pancreatitis,” Journal of the American Medical Association, vol. 285, no. 21, pp. 2716–2717, 2001.
- A. Schneider, R. H. Pfützer, M. M. Barmada, A. Slivka, J. Martin, and D. C. Whitcomb, “Limited contribution of the SPINK1 N34S mutation to the risk and severity of alcoholic chronic pancreatitis: a report from the United States,” Digestive Diseases and Sciences, vol. 48, no. 6, pp. 1110–1115, 2003.
- F. Perri, A. Piepoli, P. Stanziale, A. Merla, L. Zelante, and A. Andriulli, “Mutation analysis of the cystic fibrosis transmembrane conductance regulator (CFTR) gene, the cationic trypsinogen (PRSS1) gene, and the serine protease inhibitor, Kazal type 1 (SPINK1) gene in patientS with alcoholic chronic pancreatitis,” European Journal of Human Genetics, vol. 11, no. 9, pp. 687–692, 2003.
- E. Aoun, V. Muddana, G. I. Papachristou, and D. C. Whitcomb, “SPINK1 N34S is strongly associated with recurrent acute pancreatitis but is not a risk factor for the first or sentinel acute pancreatitis event,” American Journal of Gastroenterology, vol. 105, no. 2, pp. 446–451, 2010.
- M. Lempinen, U. H. Stenman, J. Halttunen, P. Puolakkainen, R. Haapiainen, and E. Kemppainen, “Early sequential changes in serum markers of acute pancreatitis induced by endoscopic retrograde cholangiopancreatography,” Pancreatology, vol. 5, no. 2-3, pp. 157–164, 2005.