International Journal of Endocrinology / 2018 / Article / Fig 3

Clinical Study

Newer Perspectives of Mechanisms for Euglycemic Diabetic Ketoacidosis

Figure 3

EDKA in a non-insulin-dependent setting: triggered by starvation or other precipitants; with the compensation capacity of EGP impaired by lack of substrates or poor liver function, a metabolic shift to lipid utilization occurs at a lower blood glucose concentration.

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