Research Article

and Receptors Contribute to Serotonin-Induced Vascular Dysfunction in Diabetes

Figure 2

Relaxation of aorta from non-diabetics (ND) and diabetics (DB) to serotonin with (E+) and without (E−) endothelium (a), following inhibition of nitric oxide synthase with nitro-L-arginine (LNNA, b) and LNNA with H1152 (c). All data are mean ± SEM, versus ND, versus control.
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(a)
398406.fig.002b
(b)
398406.fig.002c
(c)