and Receptors Contribute to Serotonin-Induced Vascular Dysfunction in Diabetes
Figure 2
Relaxation of aorta from non-diabetics (ND) and diabetics (DB) to serotonin with (E+) and without (E−) endothelium (a), following inhibition of nitric oxide synthase with nitro-L-arginine (LNNA, b) and LNNA with H1152 (c). All data are mean ± SEM, versus ND, versus control.