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| Kinase name | Kinase type | Splicing | Regulation type |
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| Fyn | Cytosolic tyrosine kinase | Alternative use of exon 7a or 7b upstream of the kinase domain | Kinase activity modulation by interfering with autoinhibition |
| Fak | Focal adhesion tyrosine kinase | Multiple alternative splicing upstream of the kinase domain | Kinase activity modulation by interfering with autophosphorylation |
| B-Raf | Cytosolic serine/threonine kinase | Alternatively spliced exon 8b and 9b upstream of the kinase domain | Kinase activity modulation by interfering with phosphorylation and autoinhibition |
Intact kinase domain | Ret | Membrane-bound tyrosine kinase receptor | C-terminal alternative splicing generating three isoforms | Modulation of signaling partners binding |
| ErbB4 | Membrane-bound tyrosine kinase receptor | N- and C-terminal alternative splicing generating four isoforms | Modulation of partners binding, cleavage, and subcellular localization |
| FGFR1 FGFR2 FGFR3 | Membrane-bound tyrosine kinase receptors | Alternative use of exon 8 or 9 generating distinct extracellular immunoglobulin-like domain III | Modified FGF binding specificity |
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| A-Raf | Cytosolic serine/threonine kinase | Intronic sequences retention introducing stop codons | Dominant negative |
Kinase domain truncation | TrkB TrkC | Membrane-bound tyrosine kinase receptors | C-terminal alternative splicing replacing kinase domain by short amino acid sequences | Ligand sequestering, dominant negative and/or specific signaling functions |
| VEGFR1 VEGFR2 | Membrane-bound tyrosine kinase receptors | C-terminal alternative splicing eliminating the kinase and transmembrane domains | Synthesis of secreted/soluble extracellular ligand-binding domains |
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