Secondary osteoporosis results from chronic conditions that contribute significantly to accelerated bone loss. The term “secondary” applies to patients in whom the identifiable causal factors are other than aging and menopause. Secondary osteoporosis should especially be considered and extensively investigated in children, in men at all ages, and in premenopausal women with atraumatic fractures. Beyond the well-recognized associations between the risk of secondary osteoporosis and long-term corticosteroids use, an increasing list of endocrine and metabolic disorders has also postulated them to be important factors in causing bone loss and fractures.

This will be a Special Issue on endocrine and metabolic causes of bone mass deterioration that may address current unmet needs regarding epidemiology, pathogenesis, diagnosis, and treatment of such patients. We cordially invite authors to present original research as well as review articles that will stimulate the continuing efforts in the understanding of mechanism involved in the development of secondary osteoporosis. Main topics include, but are not limited to:

• Diabetes mellitus
• Pituitary gland diseases
• Estrogen/androgen deficiency
• Hyperthyroidism
• Pregnancy
• Glucocorticoid-induced osteoporosis
• End-stage renal disease
• Organ transplantation
• Malabsorption
• Alcohol abuse
• Chronic metabolic acidosis
• Bone loss associated with chronic inflammation

Before submission authors should carefully read over the journal's Author Guidelines, which are located at http://www.hindawi.com/journals/josteo/guidelines/. Prospective authors should submit an electronic copy of their complete manuscript through the journal Manuscript Tracking System at http://mts.hindawi.com/ according to the following timetable: